Evidence map›Paper›PMID 9891065›Full record

ArticleMolecular and cellular biology1999

Novel roles of specific isoforms of protein kinase C in activation of the c-fos serum response element.

J W Soh, E H Lee, R Prywes, I B Weinstein

Open access · greenAbstract read
In one paragraph

Article in Molecular and cellular biology, 1999. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 81 papers.

0numbers the graph read from it
0cells of the map it votes in
81citing papers in PubMed
8.3field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

81 citing papers in PubMed, 258 citations in OpenAlex.

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  11. Hypothalamic extended synaptotagmin-3 contributes to the development of dietary obesity and related metabolic disorders.Proceedings of the National Academy of Sciences of the United States of America · 2020
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  19. Control of nuclear-cytoplasmic shuttling of Ankrd54 by PKCδ.World journal of biological chemistry · 2017
    Article
  20. Article

21 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

J W SohDepartment of Biochemistry & Molecular Biophysics, College of Physicians & Surgeons, Columbia University, New York, New York 10032, USA.
E H Lee
R Prywes
I B Weinstein
Columbia University · US

Funding

Transcriptional regulation of c-fos and immediate early genesR01CA050329 · NCI · COLUMBIA UNIV NEW YORK MORNINGSIDE · PI PRYWES, RON M · 1989 to 2009
$3.3M
ROLES OF SPECIFIC ISOFORMS OF PKC IN CELL CYCLE CONTROLR01CA026056 · NCI · COLUMBIA UNIV NEW YORK MORNINGSIDE · PI WEINSTEIN, I. BERNARD · 1985 to 2003
$1.1M
CELLULAR AND BIOCHEMICAL EFFECTS OF TUMOR PROMOTERSR37CA026056 · NCI · COLUMBIA UNIV NEW YORK MORNINGSIDE · PI WEINSTEIN, I. BERNARD · 1990 to 1998
–
NCI NIH HHS CA26056NCI NIH HHS CA50329NCI NIH HHS R01 CA050329
6 · The paper itself

Abstract

Protein kinase C (PKC) is a multigene family of enzymes consisting of at least 11 isoforms. It has been implicated in the induction of c-fos and other immediate response genes by various mitogens. The serum response element (SRE) in the c-fos promoter is necessary and sufficient for induction of transcription of c-fos by serum, growth factors, and the phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA). It forms a complex with the ternary complex factor (TCF) and with a dimer of the serum response factor (SRF). TCF is the target of several signal transduction pathways and SRF is the target of the rhoA pathway. In this study we generated dominant-negative and constitutively active mutants of PKC-alpha, PKC-delta, PKC-epsilon, and PKC-zeta to determine the roles of individual isoforms of PKC in activation of the SRE. Transient-transfection assays with NIH 3T3 cells, using an SRE-driven luciferase reporter plasmid, indicated that PKC-alpha and PKC-epsilon, but not PKC-delta or PKC-zeta, mediate SRE activation. TPA-induced activation of the SRE was partially inhibited by dominant negative c-Raf, ERK1, or ERK2, and constitutively active mutants of PKC-alpha and PKC-epsilon activated the transactivation domain of Elk-1. TPA-induced activation of the SRE was also partially inhibited by a dominant-negative MEKK1. Furthermore, TPA treatment of serum-starved NIH 3T3 cells led to phosphorylation of SEK1, and constitutively active mutants of PKC-alpha and PKC-epsilon activated the transactivation domain of c-Jun, a major substrate of JNK. Constitutively active mutants of PKC-alpha and PKC-epsilon could also induce a mutant c-fos promoter which lacks the TCF binding site, and they also induce transactivation activity of the SRF. Furthermore, rhoA-mediated SRE activation was blocked by dominant negative mutants of PKC-alpha or PKC-epsilon. Taken together, these findings indicate that PKC-alpha and PKC-epsilon can enhance the activities of at least three signaling pathways that converge on the SRE: c-Raf-MEK1-ERK-TCF, MEKK1-SEK1-JNK-TCF, and rhoA-SRF. Thus, specific isoforms of PKC may play a role in integrating networks of signal transduction pathways that control gene expression.

Indexed as

Genes, fosMAP Kinase Kinase 4MAP Kinase Kinase Kinase 1Mitogen-Activated Protein Kinase Kinases3T3 CellsAnimalsBase SequenceBinding SitesCOS CellsGene Expression RegulationHumansIsoenzymesMAP Kinase Kinase 1MiceMutationPlasmidsIsoenzymesMAP2K1 protein, humanMap2k1 protein, mouseMAP2K4 protein, humanMap2k4 protein, mouseMAP3K1 protein, humanMap3k1 protein, mouseMAP Kinase Kinase 1MAP Kinase Kinase 4MAP Kinase Kinase Kinase 1Mitogen-Activated Protein Kinase KinasesProtein Kinase CProtein KinasesProtein Serine-Threonine KinasesProtein-Tyrosine KinasesProto-Oncogene Proteins c-rafTetradecanoylphorbol Acetate

Identifiers

PMID9891065
PMCPMC116060
OpenAlexW2160582900

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.