ArticleAmerican journal of human genetics1998
Genome screens using linkage disequilibrium tests: optimal marker characteristics and feasibility.
Article in American journal of human genetics, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 36 papers, 1 of them a synthesis that pooled it.
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Who cites it
36 citing papers in PubMed, 1 synthesis or guideline pooled it.
- A meta-analysis strategy for gene prioritization using gene expression, SNP genotype, and eQTL data.BioMed research international · 2015Pooled it
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- Cumulative risk on the oxytocin receptor gene (OXTR) underpins empathic communication difficulties at the first stages of romantic love.Social cognitive and affective neuroscience · 2014Article
- Genome-wide linkage disequilibrium in nine-spined stickleback populations.G3 (Bethesda, Md.) · 2014Article
- GABRA2 markers moderate the subjective effects of alcohol.Addiction biology · 2013Article
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- Association of haplotypes of inflammation-related genes with gastric preneoplastic lesions in African Americans and Caucasians.International journal of cancer · 2011Article
- A Bayesian hierarchical model for detecting haplotype-haplotype and haplotype-environment interactions in genetic association studies.Human heredity · 2011Article
- A genome-wide association scan for rheumatoid arthritis data by Hotelling's T2 tests.BMC proceedings · 2009Article
- Case-control association testing in the presence of unknown relationships.Genetic epidemiology · 2009Article
- Identification of novel susceptibility loci for Guam neurodegenerative disease: challenges of genome scans in genetic isolates.Human molecular genetics · 2009Article
- Common variants in the JAZF1 gene associated with height identified by linkage and genome-wide association analysis.Human molecular genetics · 2009Article
- Methods for detecting associations with rare variants for common diseases: application to analysis of sequence data.American journal of human genetics · 2008Article
- Linkage disequilibrium between STRPs and SNPs across the human genome.American journal of human genetics · 2008Article
- Article
- Nonlinear tests for genomewide association studies.Genetics · 2006Article
- Genetic structural differences between responders and non-responders to interferon therapy for chronic hepatitis-B patients.Journal of human genetics · 2006Article
- Targeted oligonucleotide-mediated microsatellite identification (TOMMI) from large-insert library clones.BMC genetics · 2005Article
- An entropy-based statistic for genomewide association studies.American journal of human genetics · 2005Article
- Sibship T2 association tests of complex diseases for tightly linked markers.Human genomics · 2005Review
Corrections and comments
- Erratum issued
Authors and funding
2 authors.
Funding
Abstract
Linkage disequilibrium (LD) testing has become a popular and effective method of fine-scale disease-gene localization. It has been proposed that LD testing could also be used for genome screening, particularly as dense maps of diallelic markers become available and automation allows inexpensive genotyping of diallelic markers. We compare diallelic markers and multiallelic markers in terms of sample sizes required for detection of LD, by use of a single marker locus in a case-control study, for rare monophyletic diseases with Mendelian inheritance. We extrapolate from our results to discuss the feasibility of single-marker LD screening in more-complex situations. We have used a deterministic population genetic model to calculate the expected power to detect LD as a function of marker density, age of mutation, number of marker alleles, mode of inheritance of a rare disease, and sample size. Our calculations show that multiallelic markers always have more power to detect LD than do diallelic markers (under otherwise equivalent conditions) and that the ratio of the number of diallelic to the number of multiallelic markers needed for equivalent power increases with mutation age and complexity of mode of inheritance. Power equivalent to that achieved by a multiallelic screen can theoretically be achieved by use of a more dense diallelic screen, but mapping panels of the necessary resolution are not currently available and may be difficult to achieve. Genome screening that uses single-marker LD testing may therefore be feasible only for young (<20 generations), rare, monophyletic Mendelian diseases, such as may be found in rapidly growing genetic isolates.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.