Evidence map›Paper›PMID 9800206›Full record

ReviewPhilosophical transactions of the Royal Society of London. Series B, Biological sciences1998

The molecular basis of ethylene signalling in Arabidopsis.

K Woeste, J J Kieber

Abstract readReview
In one paragraph

Review in Philosophical transactions of the Royal Society of London. Series B, Biological sciences, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

K WoesteDepartment of Biological Sciences, University of Illinois at Chicago 60607, USA.
J J Kieber

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The simple gas ethylene profoundly influences plants at nearly every stage of growth and development. In the past ten years, the use of a genetic approach, based on the triple response phenotype, has been a powerful tool for investigating the molecular events that underlie these effects. Several fundamental elements of the pathway have been described: a receptor with homology to bacterial two-component histidine kinases (ETR1), elements of a MAP kinase cascade (CTR1) and a putative transcription factor (EIN3). Taken together, these elements can be assembled into a simple, linear model for ethylene signalling that accounts for most of the well-characterized ethylene mediated responses.

Indexed as

Arabidopsis ProteinsArabidopsisBase SequenceCalcium-Calmodulin-Dependent Protein KinasesDNA-Binding ProteinsDNA PrimersEthylenesModels, BiologicalMutationNuclear ProteinsPlant ProteinsReceptors, Cell SurfaceSignal TransductionTranscription FactorsArabidopsis ProteinsCalcium-Calmodulin-Dependent Protein KinasesDNA-Binding ProteinsDNA PrimersEIN3 protein, Arabidopsisethyleneethylene receptors, plantEthylenesNuclear ProteinsPlant ProteinsReceptors, Cell SurfaceTranscription FactorsNASA Discipline Plant BiologyNon-NASA Center

Identifiers

PMID9800206
PMCPMC1692355

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.