ArticleMolecular and cellular biochemistry1998
Quantitative studies of enzyme-substrate compartmentation, functional coupling and metabolic channelling in muscle cells.
Article in Molecular and cellular biochemistry, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.
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Who cites it
16 citing papers in PubMed, 48 citations in OpenAlex.
- Modular organization of cardiac energy metabolism: energy conversion, transfer and feedback regulation.Acta physiologica (Oxford, England) · 2015Review
- Early alterations of brain cellular energy homeostasis in Huntington disease models.The Journal of biological chemistry · 2012Article
- Direct measurement of energy fluxes from mitochondria into cytoplasm in permeabilized cardiac cells in situ: some evidence for Mitochondrial Interactosome.Journal of bioenergetics and biomembranes · 2009Article
- MitoKATP activity in healthy and ischemic hearts.Journal of bioenergetics and biomembranes · 2009Review
- Philosophical basis and some historical aspects of systems biology: from Hegel to Noble - applications for bioenergetic research.International journal of molecular sciences · 2009Review
- Heterogeneity of ADP diffusion and regulation of respiration in cardiac cells.Biophysical journal · 2003Article
- An integrated model of cardiac mitochondrial energy metabolism and calcium dynamics.Biophysical journal · 2003Article
- Mitochondria do not control heart bioenergetics.Molecular biology reports · 2002Article
- Adenylate kinase 1 gene deletion disrupts muscle energetic economy despite metabolic rearrangement.The EMBO journal · 2000Article
- Developmental changes in regulation of mitochondrial respiration by ADP and creatine in rat heart in vivo.Molecular and cellular biochemistry · 2000Article
- On the origin of intracellular compartmentation and organized metabolic systems.Molecular and cellular biochemistryReview
- Two structurally distinct and spatially compartmentalized adenylate kinases are expressed from the AK1 gene in mouse brain.Molecular and cellular biochemistryArticle
- Structural and functional adaptations of striated muscles to CK deficiency.Molecular and cellular biochemistryReview
- CK flux or direct ATP transfer: versatility of energy transfer pathways evidenced by NMR in the perfused heart.Molecular and cellular biochemistryReview
- Alterations of the bioenergetics systems of the cell in acute and chronic myocardial ischemia.Molecular and cellular biochemistryArticle
- Functional coupling as a basic mechanism of feedback regulation of cardiac energy metabolism.Molecular and cellular biochemistryArticle
Corrections and comments
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Authors and funding
4 authors at 4 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Some historical aspects of development of the concepts of functional coupling, metabolic channelling, compartmentation and energy transfer networks are reviewed. Different quantitative approaches, including kinetic and mathematical modeling of energy metabolism, intracellular energy transfer and metabolic regulation of energy production and fluxes in the cells in vivo are analyzed. As an example of the system with metabolic channelling, thermodynamic aspects of the functioning the mitochondrial creatine kinase functionally coupled to the oxidative phosphorylation are considered. The internal thermodynamics of the mitochondrial creatine kinase reaction is similar to that for other isoenzymes of creatine kinase, and the oxidative phosphorylation process specifically influences steps of association and dissociation of MgATP with the enzyme due to channelling of ATP from adenine nucleotide translocase. A new paradigm of muscle bioenergetics-the paradigm of energy transfer and feedback signaling networks based on analysis of compartmentation phenomena and structural and functional interactions in the cell is described. Analysis of the results of mathematical modeling of the compartmentalized energy transfer leads to conclusion that both calcium and ADP, which concentration changes synchronously in contraction cycle, may simultaneously activate oxidative phosphorylation in the muscle cells in vivo. The importance of the phosphocreatine circuit among other pathways of intracellular energy transfer network is discussed on the basis of the recent data published in the literature, with some experimental demonstration. The results of studies of perfused rat hearts with completely inhibited creatine kinase show significantly decreased work capacity and respectively, energy fluxes, in these hearts in spite of significant activation of adenylate kinase system (Dzeja et al. this volume). These results, combined with those of mathematical analysis of the energy metabolism of hearts of transgenic mice with switched off creatine kinase isoenzymes confirm the importance of phosphocreatine pathway for energy transfer for cell function and energetics in mature heart and many other types of cells, as one of major parts of intracellular energy transfer network and metabolic regulation.
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9746326W1564575810What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.