Evidence map›Paper›PMID 9637706›Full record

ArticleThe Journal of clinical investigation1998

beta2-Microglobulin mutations, HLA class I antigen loss, and tumor progression in melanoma.

D J Hicklin, Z Wang, F Arienti, L Rivoltini, G Parmiani, S Ferrone

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of clinical investigation, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 62 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
62citing papers in PubMed, 1 pooled it
4.4field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

62 citing papers in PubMed, 1 synthesis or guideline pooled it, 197 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Article
  4. Article
  5. Proceedings of the National Academy of Sciences of the United States of America · 2025
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  14. Article
  15. Article
  16. Tumor microenvironment of adult T-cell leukemia/lymphoma.Journal of clinical and experimental hematopathology : JCEH · 2021
    Review
  17. Overcoming Immune Evasion in Melanoma.International journal of molecular sciences · 2020
    Review
  18. Review
  19. Review
  20. Biological Factors behind Melanoma Response to Immune Checkpoint Inhibitors.International journal of molecular sciences · 2020
    Review

2 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 2 countries.

D J HicklinDepartment of Microbiology and Immunology, New York Medical College, Valhalla, New York 10595, USA.
Z Wang
F Arienti
L Rivoltini
G Parmiani
S Ferrone
Fondazione IRCCS Istituto Nazionale dei Tumori · ITNew York Medical College · US

Funding

MONOCLONAL ANTIBODIES TO HUMAN MELANOMA ANTIGENSR01CA037959 · NCI · ROSWELL PARK CANCER INSTITUTE CORP · PI FERRONE, SOLDANO · 1985 to 2001
$518k
IMMUNOTHERAPY OF MELANOMA WITH ANTIIDIOTYPIC ANTIBODIESR01CA051814 · NCI · ROSWELL PARK CANCER INSTITUTE CORP · PI FERRONE, SOLDANO · 1991 to 1999
–
TUMOR CELL INTERACTION WITH VASCULAR CELLSR29CA067988 · NCI · SCRIPPS RESEARCH INSTITUTE · PI FELDING-HABERMANN, BRUNHILDE · 1995 to 1999
–
NCI NIH HHS CA37959NCI NIH HHS CA51814NCI NIH HHS CA67988
6 · The paper itself

Abstract

The potential negative impact of HLA class I antigen abnormalities on the outcome of T cell-based immunotherapy of melanoma has prompted us to investigate the mechanisms underlying lack of HLA class I antigen expression by melanoma cell lines Me18105, Me9923, and Me1386. Distinct mutations in the beta2-microglobulin (beta2m) gene were identified in each cell line which result in loss of functional beta2m. In Me18105 cells, an aberrant splicing mechanism caused by an A--> G point mutation in the splice acceptor site of intron 1 of the beta2m gene, deletes 11 bp from the beta2m mRNA creating a shift in the reading frame. In Me9923 cells a 14-bp deletion in exon 2 and in Me1386 cells a CT deletion in exon 1 of the beta2m gene produce a frameshift mutation. The beta2m gene mutations identified in Me18105, Me9923, and Me1386 cells were also detected in the surgically removed melanoma lesions from which the cell lines originated. Transfection of each melanoma cell line with a wild-type beta2m gene restored HLA class I antigen expression and, in Me18105 cells, recognition by Melan-A/MART-1-specific, HLA-A2-restricted cytotoxic T lymphocytes. Interestingly, the beta2m mutation present in Me9923 cells that were derived from a metastatic lesion was also found in the Me9923P cell line that originated from the autologous primary lesion. These data suggest that beta2m mutations in melanoma cells may be an early event in progression to the malignant phenotype.

Indexed as

Amino Acid SequenceBase Sequencebeta 2-MicroglobulinCell DivisionFrameshift MutationGene Expression Regulation, NeoplasticHistocompatibility Antigens Class IHumansMelanomaMolecular Sequence DataPoint MutationRNA SplicingTransfectionTumor Cells, CulturedTumor Escapebeta 2-MicroglobulinHistocompatibility Antigens Class I

Identifiers

PMID9637706
PMCPMC508863
OpenAlexW2066960417

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.