ReviewClinical cardiology1998
Monocyte-endothelial cell interaction in atherogenesis and thrombosis.
Review in Clinical cardiology, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
8 citing papers in PubMed, 39 citations in OpenAlex.
- Long non-coding RNA LINC00607 epigenetically regulates endothelial TSPAN18 to promote hypoxia-induced thromboinflammation.The Journal of biological chemistry · 2026Article
- Pathogenic Stress Induces Human Monocyte to Express an Extracellular Web of Tunneling Nanotubes.Frontiers in immunology · 2021Article
- Article
- Oxidative stress accelerates the carotid atherosclerosis process in patients with chronic kidney disease.Archives of medical sciences. Atherosclerotic diseases · 2020Article
- Increased monocyte-derived reactive oxygen species in type 2 diabetes: role of endoplasmic reticulum stress.Experimental physiology · 2017Article
- DEP induction of ROS in capillary-like endothelial tubes leads to VEGF-A expression.Toxicology · 2012Article
- Matrix metalloproteinases and atherosclerosis.Current atherosclerosis reports · 2004Review
- Matrix metalloproteinases and coronary artery diseases.Clinical cardiology · 2003Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Cellular interactions between leukocytes and the endothelium are critical events in vascular biology, such as atherosclerosis and acute coronary syndrome. When monocytes and endothelial cells are activated via direct cell-cell interaction, both types of cells express several biologically active molecules such as adhesion molecules, cytokines, coagulation and fibrinolytic factors, metalloproteinases, and vasoactive substances. All of these molecules could contribute to atherogenesis and thrombosis.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.