ReviewWorld journal of urology1997
Genes in idiopathic calcium oxalate stone disease.
Review in World journal of urology, 1997. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
17 citing papers in PubMed, 40 citations in OpenAlex.
- Etiologic risk factors and vitamin D receptor gene polymorphisms in under one-year-old infants with urolithiasis.Urolithiasis · 2018Trial
- Oxalate Alters Cellular Bioenergetics, Redox Homeostasis, Antibacterial Response, and Immune Response in Macrophages.Frontiers in immunology · 2021Article
- Sex differences in redox homeostasis in renal disease.Redox biology · 2020Review
- Inhibitive Effects of Huashi Pill on Formation of Renal Stones by Modulating Urine Biochemical Indexes and Osteopontin in Renal Stone Rat Models.Medical science monitor : international medical journal of experimental and clinical research · 2019Article
- Oxalate induces mitochondrial dysfunction and disrupts redox homeostasis in a human monocyte derived cell line.Redox biology · 2018Article
- Article
- Interstitial calcinosis in renal papillae of genetically engineered mouse models: relation to Randall's plaques.Urolithiasis · 2015Review
- Heritability of urinary traits that contribute to nephrolithiasis.Clinical journal of the American Society of Nephrology : CJASN · 2014Article
- Catechin prevents the calcium oxalate monohydrate induced renal calcium crystallization in NRK-52E cells and the ethylene glycol induced renal stone formation in rat.BMC complementary and alternative medicine · 2013Article
- Meta-analysis of the urokinase gene 3'-UTR T/C polymorphism and susceptibility to urolithiasis.Biomedical reports · 2013Article
- Calcium-sensing receptor and calcium kidney stones.Journal of translational medicine · 2011Review
- Hypocitraturia: pathophysiology and medical management.Reviews in urology · 2009Article
- Update on the genetics of nephrolithiasis.Clinical cases in mineral and bone metabolism : the official journal of the Italian Society of Osteoporosis, Mineral Metabolism, and Skeletal Diseases · 2008Article
- Predisposition of genetic polymorphism with the risk of urolithiasis.Indian journal of clinical biochemistry : IJCB · 2008Article
- Association of vitamin-D and calcitonin receptor gene polymorphism in paediatric nephrolithiasis.Pediatric nephrology (Berlin, Germany) · 2005Article
- Calcium oxalate stones in feline littermates.Journal of feline medicine and surgery · 2000Article
- Microbial relatives of the seed storage proteins of higher plants: conservation of structure and diversification of function during evolution of the cupin superfamily.Microbiology and molecular biology reviews : MMBR · 2000Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
4 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
An examination of the urinary excretions of 101 normal subjects indicated that the major genetic influence on calcium excretion is a codominant pair of alleles giving rise to three phenotypes, low, intermediate and high (hypercalciuric) excretors. This inference was based on variance, Hardy-Weinberg and segregation analyses. Similar independent gene pairs also appear to influence oxalate and citrate excretion, A 3-locus Hardy-Weinberg table using estimates of gene frequencies derived from the study of normals suggests that only 3 or 4 leading genes are involved in oxalate stone disease. Strong candidate genes identified from molecular and physiological studies cannot be proposed at present, but it is assumed that they influence the transport of these ions in either the intestine, kidney or both organs. The identification of the genes involved should be facilitated by the reduction of dietary influences on urinary excretions through the use of formula diets.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.