ReviewDrugs1997
Recognition and management of bacterial arthritis.
Review in Drugs, 1997. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
7 citing papers in PubMed, 24 citations in OpenAlex.
- Real time monitoring of Staphylococcus aureus biofilm sensitivity towards antibiotics with isothermal microcalorimetry.PloS one · 2022Article
- Acute hematogenous septic arthritis of the knee in adults.European journal of orthopaedic surgery & traumatology : orthopedie traumatologie · 2013Article
- Results of treatment of septic knee arthritis: a retrospective series of 40 cases.Knee surgery, sports traumatology, arthroscopy : official journal of the ESSKA · 2007Article
- Prospective comparative study of patients with culture proven and high suspicion of adult onset septic arthritis.Annals of the rheumatic diseases · 2003Article
- Rheumatology: 4. Acute monoarthritis.CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne · 2000Article
- Mechanisms of internalization of Staphylococcus aureus by cultured human osteoblasts.Infection and immunity · 1999Article
- Bacterial arthritis in the elderly: an overview.Drugs & aging · 1999Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Bacterial arthritis is a bacterial infection of the joint. Apart from the classical gonococcal arthritis, nongonococcal arthritides include specific forms such as mycobacterial or Borrelia burgdorferi arthritis. Almost any bacterium can cause arthritis, provided that the route of penetration and the host response are suitable. Weakening of the host's immune competence, pre-existing joint damage and invasive diagnostic or therapeutic procedures are the main risk factors for bacterial arthritis. Gram-positive cocci are the species most frequently involved. The pathogenesis of bacterial damage includes release of toxins, cell production of cytokines and autoimmune reactions to specific antigens. The diagnosis can be suspected clinically put must be confirmed by culture of the synovial fluid, a test which can be complemented by scintigraphy. Amplification of bacterial DNA by polymerase chain reaction is a new procedure that could become an important tool for quick diagnosis. Treatment is based on joint drainage and antibiotics, which should be started as soon as the diagnosis is suspected. Corollary strategies under investigation include corticosteroids to prevent joint damage, monoclonal antibodies to arthritogenic peptides of bacteria or to surface markets of host lymphocytes, and modulators of synovial fluid cytokines.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.