Ehlers-Danlos syndrome has varied molecular mechanisms.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

9 citing papers in PubMed.

1. Multiple dermatofibromas in a patient with Ehlers-Danlos syndrome: a case report.Journal of medical case reports · 2024
   Article
2. Atypical COL3A1 variants (glutamic acid to lysine) cause vascular Ehlers-Danlos syndrome with a consistent phenotype of tissue fragility and skin hyperextensibility.Genetics in medicine : official journal of the American College of Medical Genetics · 2019
   Article
3. Vascular wall extracellular matrix proteins and vascular diseases.Biochimica et biophysica acta · 2014
   Review
4. Metalloproteases meprin α and meprin β are C- and N-procollagen proteinases important for collagen assembly and tensile strength.Proceedings of the National Academy of Sciences of the United States of America · 2013
   Article
5. Matrix metalloproteinases and descending aortic aneurysms: parity, disparity, and switch.Journal of cardiac surgery · 2012
   Review
6. Recent advances in the use of serological bone formation markers to monitor callus development and fracture healing.Critical reviews in eukaryotic gene expression · 2010
   Review
7. Ehlers-Danlos syndrome with severe early-onset periodontal disease (EDS-VIII) is a distinct, heterogeneous disorder with one predisposition gene at chromosome 12p13.American journal of human genetics · 2003
   Article
8. A point mutation in an intronic branch site results in aberrant splicing of COL5A1 and in Ehlers-Danlos syndrome type II in two British families.American journal of human genetics · 1998
   Article
9. Lumican regulates collagen fibril assembly: skin fragility and corneal opacity in the absence of lumican.The Journal of cell biology · 1998
   Article

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

Authors and funding

2 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

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