Evidence map›Paper›PMID 9041695›Full record

ReviewThe Yale journal of biology and medicine

Helicobacter pylori and its interaction with chief and parietal cells.

D R Cave

Open access · greenAbstract readReview
In one paragraph

Review in The Yale journal of biology and medicine. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 8 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

D R CaveSt. Elizabeth's Medical Center, Boston, Massachusetts 02135, USA.
St. Elizabeth's Medical Center · US

Funding

REGULATION OF NET HEPATIC GLUCOSE UPTAKE IN VIVOR01DK043706 · NIDDK · VANDERBILT UNIVERSITY · PI CHERRINGTON, ALAN D · 1991 to 2009
$4.6M
NIDDK NIH HHS NIDDK 043706
6 · The paper itself

Abstract

Helicobacter pylori has been shown, along with other Helicobacter species to produce effector molecules that induce substantial physiological changes on acid and pepsin secretion. The effects are clinically evident, and long-term achlorhydria may be a risk factor for gastric cancer. Further identification and characterization of the factors may lead to additional understanding of gastric physiology.

Indexed as

AchlorhydriaAnimalsHelicobacter InfectionsHelicobacter pyloriHumansParietal Cells, Gastric

Identifiers

PMID9041695
PMCPMC2588978
OpenAlexW2415870111

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.