ArticleGenetics1996
Epistatic control of non-Mendelian inheritance in mouse interspecific crosses.
Article in Genetics, 1996. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
23 citing papers in PubMed, 76 citations in OpenAlex.
- Parent-offspring genotyped trios unravelling genomic regions with gametic and genotypic epistatic transmission bias on the cattle genome.Frontiers in genetics · 2023Article
- Meiotic drive in house mice: mechanisms, consequences, and insights for human biology.Chromosome research : an international journal on the molecular, supramolecular and evolutionary aspects of chromosome biology · 2022Review
- Craniomandibular form and body size variation of first generation mouse hybrids: A model for hominin hybridization.Journal of human evolution · 2018Article
- Review
- R2d2 Drives Selfish Sweeps in the House Mouse.Molecular biology and evolution · 2016Article
- A multi-megabase copy number gain causes maternal transmission ratio distortion on mouse chromosome 2.PLoS genetics · 2015Article
- Article
- A RAD-based linkage map and comparative genomics in the gudgeons (genus Gnathopogon, Cyprinidae).BMC genomics · 2013Article
- Article
- Systematic detection of epistatic interactions based on allele pair frequencies.PLoS genetics · 2012Article
- Genetic mapping and developmental timing of transmission ratio distortion in a mouse interspecific backcross.BMC genetics · 2010Article
- A new standard genetic map for the laboratory mouse.Genetics · 2009Article
- Mouse consomic strains: exploiting genetic divergence between Mus m. musculus and Mus m. domesticus subspecies.Genome research · 2008Article
- Article
- A large-sample QTL study in mice: III. Reproduction.Mammalian genome : official journal of the International Mammalian Genome Society · 2004Article
- Genetic analysis of X-linked hybrid sterility in the house mouse.Mammalian genome : official journal of the International Mammalian Genome Society · 2004Article
- Article
- Nonrandom segregation of the mouse univalent X chromosome: evidence of spindle-mediated meiotic drive.Genetics · 2000Article
- A genetic test to determine the origin of maternal transmission ratio distortion. Meiotic drive at the mouse Om locus.Genetics · 2000Article
- Genetic dissection of X-linked interspecific hybrid placental dysplasia in congenic mouse strains.Genetics · 1999Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors at 2 institutions in 2 countries.
Funding
Abstract
Strong deviation of allele frequencies from Mendelian inheritance favoring Mus spretus-derived alleles has been described previously for X-linked loci in four mouse interspecific crosses. We reanalyzed data for three of these crosses focusing on the location of the gene(s) controlling deviation on the X chromosome and the genetic basis for incomplete deviation. At least two loci control deviation on the X chromosome, one near Xist (the candidate gene controlling X inactivation) and the other more centromerically located. In all three crosses, strong epistasis was found between loci near Xist and marker loci on the central portion of chromosome 2. The mechanism for this deviation from Mendelian expectations is not yet known but it is probably based on lethality of embryos carrying particular combinations of alleles rather than true segregation distortion during oogenesis in F1 hybrid females.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.