ArticleThe Journal of physiology1996
Caffeine enhances intramembranous charge movement in frog skeletal muscle by increasing cytoplasmic Ca2+ concentration.
Article in The Journal of physiology, 1996. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.
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Who cites it
14 citing papers in PubMed, 24 citations in OpenAlex.
- Voltage modulates halothane-triggered CaThe Journal of general physiology · 2018Article
- Reciprocal dihydropyridine and ryanodine receptor interactions in skeletal muscle activation.Journal of muscle research and cell motility · 2011Review
- Caffeine - a valuable tool in excitation-contraction coupling research.The Journal of physiology · 2008Article
- Differential sensitivity to perchlorate and caffeine of tetracaine-resistant Ca2+ release in frog skeletal muscle.Journal of muscle research and cell motility · 2006Article
- Altered elementary calcium release events and enhanced calcium release by thymol in rat skeletal muscle.Biophysical journal · 2004Article
- Effects of oxidation and cytosolic redox conditions on excitation-contraction coupling in rat skeletal muscle.The Journal of physiology · 2003Article
- Intramembrane charge movement and L-type calcium current in skeletal muscle fibers isolated from control and mdx mice.Biophysical journal · 2003Article
- Caffeine-induced immobilization of gating charges in isolated guinea-pig ventricular heart cells.British journal of pharmacology · 2002Article
- Spark- and ember-like elementary Ca2+ release events in skinned fibres of adult mammalian skeletal muscle.The Journal of physiology · 2001Article
- Charge movements in intact amphibian skeletal muscle fibres in the presence of cardiac glycosides.The Journal of physiology · 2001Article
- Modification of excitation-contraction coupling by 4-chloro-m-cresol in voltage-clamped cut muscle fibres of the frog (R. pipiens).The Journal of physiology · 1999Article
- Caffeine and excitation-contraction coupling in skeletal muscle: a stimulating story.Journal of muscle research and cell motility · 1999Review
- The influence of caffeine on intramembrane charge movements in intact frog striated muscle.The Journal of physiology · 1998Article
- Activation of Ca2+ release by caffeine and voltage in frog skeletal muscle.The Journal of physiology · 1996Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
1. Currents of intramembranous charge movement were recorded, together with intracellular [Ca2+], in single muscle fibres subjected to voltage-clamp depolarization and 'pulses' of extracellular solution with a Ca2+ release-inducing concentration of caffeine (10 mM). 2. When caffeine was present prior to and during the voltage pulses, the charge transferred by pulses to between -60 and -40 mV increased by about 40%. 3. In fibres depleted of Ca2+ in the sarcoplasmic reticulum (SR), caffeine had no effect on charge transfer or kinetics. 4. Whenever the prior exposure to caffeine resulted in a large elevation in [Ca2+]i at the start of the depolarizing pulse, there was an increase in I beta, the monotonically decaying component of charge movement. When the presence of caffeine enhanced Ca2+ release induced by the pulse, there was increase in I gamma, the hump-like component. 5. The charge transferred during a pulse to -50 mV increased with time of exposure to caffeine. Ca2+ release induced by the voltage pulse grew during the first second of caffeine exposure, then decreased with longer exposure time. The enhancement of charge transfer by caffeine was therefore not due to the increase in Ca2+ release caused by the drug. 6. The increase in charge transfer was a uniform, monotonically increasing function of the [Ca2+]i attained at the end of the voltage pulse. 7. Charge transfer, as a function of [Ca2+]i, pulse voltage and time, was simulated with a model, used previously, in which Ca2+ binds to intracellular sites and increases the electrical potential near the voltage sensors. Two sites were needed to fit the observations, with dissociation constants of 60 nM and 2 to 10 microM. 8. In the presence of caffeine, the voltage-driven movement of a given amount of intra-membranous charge resulted in greater activation of release permeability. 9. All effects of caffeine observed in this and the preceding paper could be explained assuming a single action: caffeine increases the tendency of the release channels to open. This results in opening of closed channels and an increase in their susceptibility to activation by the voltage sensors.
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