ReviewMolecular and cellular biochemistry
Signalling by protein kinase C isoforms in the heart.
Review in Molecular and cellular biochemistry. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
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Who cites it
11 citing papers in PubMed, 81 citations in OpenAlex.
- Protein kinase C and cardiac dysfunction: a review.Heart failure reviews · 2017Review
- Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice.The Journal of clinical investigation · 2012Article
- Perinatal asphyxia: current status and approaches towards neuroprotective strategies, with focus on sentinel proteins.Neurotoxicity research · 2011Review
- AMP-activated protein kinase activation as a strategy for protecting vascular endothelial function.Clinical and experimental pharmacology & physiology · 2008Review
- VEGF-PLCgamma1 pathway controls cardiac contractility in the embryonic heart.Genes & development · 2005Article
- Defective sarcolemmal phospholipase C signaling in diabetic cardiomyopathy.Molecular and cellular biochemistry · 2004Article
- G-proteins in growth and apoptosis: lessons from the heart.Oncogene · 2001Review
- Molecular and cellular mechanisms of cardiotoxicity.Environmental health perspectives · 2001Review
- Endothelin-1 and photoreleased diacylglycerol increase L-type Ca2+ current by activation of protein kinase C in rat ventricular myocytes.The Journal of physiology · 2000Article
- Activation of epsilon protein kinase C correlates with a cardioprotective effect of regular ethanol consumption.Proceedings of the National Academy of Sciences of the United States of America · 1998Article
- Losartan attenuates phospholipase C isozyme gene expression in hypertrophied hearts due to volume overload.Journal of cellular and molecular medicineArticle
Corrections and comments
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Understanding transmembrane signalling process is one of the major challenge of the decade. In most tissues, since Fisher and Krebs's discovery in the 1950's, protein phosphorylation has been widely recognized as a key event of this cellular function. Indeed, binding of hormones or neurotransmitters to specific membrane receptors leads to the generation of cytosoluble second messengers which in turn activate a specific protein kinase. Numerous protein kinases have been so far identified and roughly classified into two groups, namely serine/threonine and tyrosine kinases on the basis of the target acid although some more recently discovered kinases like MEK (or MAP kinase kinase) phosphorylate both serine and tyrosine residues. Protein kinase C is a serine/threonine kinase that was first described by Takai et al. [1] as a Ca- and phospholipid-dependent protein kinase. Later on, Kuo et al. [2] found that PKC was expressed in most tissues including the heart. The field of investigation became more complicated when it was found that the kinase is not a single molecular entity and that several isoforms exist. At present, 12 PKC isoforms and other PKC-related kinases [3] were identified in mammalian tissues. These are classified into three groups. (1) the Ca-activated alpha-, beta-, and gamma-PKCs which display a Ca-binding site (C2); (2) the Ca-insensitive delta-, epsilon-, theta-, eta-, and mu-PKCs. The kinases that belong to both of these groups display two cysteine-rich domains (C1) which bind phorbol esters (for recent review on PKC structure, see [4]). (3) The third group was named atypical PKCs and include zeta, lambda, and tau-PKCs that lack both the C2 and one cysteine-rich domain. Consequently, these isoforms are Ca-insensitive and cannot be activated by phorbol esters [5]. In the heart, evidence that multiple PKC isoforms exist was first provided by Kosaka et at. [6] who identified by chromatography at least two PKC-related isoenzymes. Numerous studies were thus devoted to the biochemical characterization of these isoenzymes (see [7] for review on cardiac PKCs) as well as to the identification of their substrates. This overview aims at updating the present knowledge on the expression, activation and functions of PKC isoforms in cardiac cells.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.