ArticleJapanese journal of cancer research : Gann1996
A quantitative trait locus in major histocompatibility complex determining latent period of mouse lymphomas.
Article in Japanese journal of cancer research : Gann, 1996. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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Who cites it
3 citing papers in PubMed.
- Pre-B lymphomas in SL/Kh mice: a multifactorial disease model.Cancer science · 2003Review
- Propylnitrosourea-induced T-lymphomas in LEXF RI strains of rats: genetic analysis.British journal of cancer · 1999Article
- Quantitative trait loci associated with promoting effects of sodium L-ascorbate on two-stage bladder carcinogenesis in rats.Japanese journal of cancer research : Gann · 1997Article
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Authors and funding
7 authors.
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Abstract
The effects of two host genes on retrovirus-induced murine lymphoma were evaluated by studying 114 F2 intercross mice between SL/Kh and AKR/Ms mice. Out of 47 T-lymphoma-bearing F2 mice, 45 had the AKR-derived dominant allele at Tism-1. The length of the lymphoma latent period was not related to type of tumor. Instead, it was significantly shortened by a recessive SL/Kh-derived allele at a major histocompatibility complex (MHC)-linked locus on Chr. 17. A quantitative trait analysis of the latent period yielded a maximal logarithm of likelihood ratio for linkage (LOD) score of 7.06 at a class II gene within MHC. The SL/Kh-derived recessive gene was named lla (lymphoma latency acceleration).
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