ArticleGenetics1995
Molecular analysis of 36 mutations at the mouse pink-eyed dilution (p) locus.
Article in Genetics, 1995. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
23 citing papers in PubMed, 68 citations in OpenAlex.
- Causal gene identification and desirable trait recreation in goldfish.Science China. Life sciences · 2022Article
- A novel porcine model reproduces human oculocutaneous albinism type II.Cell discovery · 2019Article
- The neurobiology of mouse models syntenic to human chromosome 15q.Journal of neurodevelopmental disorders · 2011Article
- Creating a "hopeful monster": mouse forward genetic screens.Methods in molecular biology (Clifton, N.J.) · 2011Article
- Predicting phenotype from genotype: normal pigmentation.Journal of forensic sciences · 2010Article
- Article
- Mouse chromosome engineering for modeling human disease.Annual review of genomics and human genetics · 2006Review
- X-ray-induced deletion complexes in embryonic stem cells on mouse chromosome 15.Mammalian genome : official journal of the International Mammalian Genome Society · 2005Article
- Lack of Pwcr1/MBII-85 snoRNA is critical for neonatal lethality in Prader-Willi syndrome mouse models.Mammalian genome : official journal of the International Mammalian Genome Society · 2005Article
- Cloning, functional study and comparative mapping of Luzp2 to mouse chromosome 7 and human chromosome 11p13-11p14.Mammalian genome : official journal of the International Mammalian Genome Society · 2003Article
- Evidence for the role of PWCR1/HBII-85 C/D box small nucleolar RNAs in Prader-Willi syndrome.American journal of human genetics · 2002Article
- Functional annotation of mammalian genomic DNA sequence by chemical mutagenesis: a fine-structure genetic mutation map of a 1- to 2-cM segment of mouse chromosome 7 corresponding to human chromosome 11p14-p15.Proceedings of the National Academy of Sciences of the United States of America · 2002Article
- Distinct phenotypes distinguish the molecular classes of Angelman syndrome.Journal of medical genetics · 2001Article
- Molecular and functional mapping of the piebald deletion complex on mouse chromosome 14.Genetics · 2001Article
- Small evolutionarily conserved RNA, resembling C/D box small nucleolar RNA, is transcribed from PWCR1, a novel imprinted gene in the Prader-Willi deletion region, which Is highly expressed in brain.American journal of human genetics · 2000Article
- Interdigitated deletion complexes on mouse chromosome 5 induced by irradiation of embryonic stem cells.Genome research · 2000Article
- Sox6 is a candidate gene for p100H myopathy, heart block, and sudden neonatal death.Proceedings of the National Academy of Sciences of the United States of America · 2000Article
- Identification of novel imprinted transcripts in the Prader-Willi syndrome and Angelman syndrome deletion region: further evidence for regional imprinting control.American journal of human genetics · 2000Article
- Genetics of Angelman syndrome.American journal of human genetics · 1999Review
- Differential response of mouse male germ-cell stages to radiation-induced specific-locus and dominant mutations.Genetics · 1998Article
Corrections and comments
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Authors and funding
6 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Thirty-six radiation- or chemically induced homozygous-lethal mutations at the p locus in mouse chromosome 7 have been analyzed at 17 loci defined by molecular probes to determine the types of lesions, numbers of p-region markers deleted or rearranged, regions of overlap of deletion mutations, and genetic distances between loci. A linear deletion map of the [Myod1, Ldh3]-[Snrpn, Znf127] region has been constructed from the molecular analyses of the p-locus deletions. The utility of these deletions as tools for the isolation and characterization of the genes specifying the neurological, reproductive, and developmental phenotypes genetically mapped to this region will grow as more detailed molecular analyses continue.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.