Evidence map›Paper›PMID 8595655›Full record

ArticleCurrent genetics1995

The orientation of gene maps by recombination of flanking markers for the am locus of Neurospora crassa.

F J Bowring, D E Catcheside

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Article in Current genetics, 1995. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

F J BowringSchool of Biological Sciences, Flinders University, Bedford Park, South Australia 5042.
D E Catcheside

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Fincham (1967), Smyth (1973 b) and Rambosek and Kinsey (1983) have each generated fine-structure maps of the am gene of Neurospora crassa. Each map had a consistent linear order of alleles but the assignment of an orientation with respect to other linkage group-V loci differed. Fincham found the end marked by the am6 allele to be at the distal end of the locus, Smyth found am6 to be at the proximal end while the data of Rambosek and Kinsey did not suggest an orientation. Smyth s orientation has been adopted as the standard, but not unreservedly. We have aligned the genetic and physical maps of the am gene, showing that am6 is at the distal end, supporting Fincham's orientation. However, we suggest that an assumption used to orient fine structure genetic maps is flawed and that the conflicting orientation between these three studies follows from the different choice of flanking markers.

Indexed as

CinnamatesCytoskeletal ProteinsMAP Kinase Kinase KinasesRecombination, GeneticRNA, UntranslatedChromosome MappingChromosomes, FungalCrosses, GeneticDrug Resistance, MicrobialHygromycin BNeoplasm ProteinsNeurospora crassaPolymorphism, Restriction Fragment LengthProteinsProtein Serine-Threonine KinasesProto-Oncogene Proteinsactin-modulating proteinsCinnamatesCytoskeletal Proteinshygromycin AHygromycin BMAP Kinase Kinase KinasesNeoplasm ProteinsProteinsProtein Serine-Threonine KinasesProto-Oncogene ProteinsRNA, Long NoncodingRNA, UntranslatedVis1 long non-coding RNA, mouse

Identifiers

PMID8595655

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.