ArticleJournal of virology1996
Vaccinia virions lacking core protein VP8 are deficient in early transcription.
Article in Journal of virology, 1996. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.
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Who cites it
23 citing papers in PubMed, 31 citations in OpenAlex.
- Lumpy Skin Disease Virus Pathogenesis: Viral Protein Functions and Comparative Insights from Vaccinia Virus.Animals : an open access journal from MDPI · 2025Review
- Assembly and Evolution of Poxviruses.Advances in experimental medicine and biology · 2024Review
- The Vaccinia virion: Filling the gap between atomic and ultrastructure.PLoS pathogens · 2019Article
- Article
- Vaccinia Virus B1 Kinase Is Required for Postreplicative Stages of the Viral Life Cycle in a BAF-Independent Manner in U2OS Cells.Journal of virology · 2015Article
- Fine structure of the vaccinia virion determined by controlled degradation and immunolocalization.Virology · 2015Article
- Vaccinia virus mutations in the L4R gene encoding a virion structural protein produce abnormal mature particles lacking a nucleocapsid.Journal of virology · 2014Article
- Article
- Attenuation of vaccinia virus by the expression of human Flt3 ligand.Virology journal · 2010Article
- Temperature-sensitive mutant in the vaccinia virus E6 protein produce virions that are transcriptionally inactive.Virology · 2010Article
- The vaccinia virus E8R gene product is required for formation of transcriptionally active virions.Virology · 2007Article
- Vaccinia virus A12L protein and its AG/A proteolysis play an important role in viral morphogenic transition.Virology journal · 2007Article
- The conserved poxvirus L3 virion protein is required for transcription of vaccinia virus early genes.Journal of virology · 2005Article
- Vaccinia virus entry into cells is dependent on a virion surface protein encoded by the A28L gene.Journal of virology · 2004Article
- Endoplasmic reticulum-Golgi intermediate compartment membranes and vimentin filaments participate in vaccinia virus assembly.Journal of virology · 2002Article
- Microtubule-dependent organization of vaccinia virus core-derived early mRNAs into distinct cytoplasmic structures.Molecular biology of the cell · 2001Article
- The major core protein P4a (A10L gene) of vaccinia virus is essential for correct assembly of viral DNA into the nucleoprotein complex to form immature viral particles.Journal of virology · 2001Article
- Genome-wide analysis of vaccinia virus protein-protein interactions.Proceedings of the National Academy of Sciences of the United States of America · 2000Article
- Elucidating the essential role of the A14 phosphoprotein in vaccinia virus morphogenesis: construction and characterization of a tetracycline-inducible recombinant.Journal of virology · 2000Article
- Virions of Heliothis armigera entomopoxvirus contain a homologue of the vaccinia VP8 major core protein.Virus genes · 1999Article
Corrections and comments
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
When synthesis of the 25-kDa vaccinia virus core protein VP8 is repressed, mature virus particles of normal appearance are produced to approximately 80% of wild-type levels but these particles are over 100-fold less infectious than wild-type particles (D. Wilcock and G. L. Smith, Virology 202:294-304, 1994). Here we show that virions which lack VP8 can bind to and enter cells but the levels of steady-state RNA are greatly reduced in comparison with those for wild-type infections. In vitro assays using permeabilized virions demonstrated that VP8-deficient virions had drastically reduced rates of transcription (RNA synthesis was decreased by 80 to 96%) and that the extrusion of RNA transcripts from these virions was also decreased. Low concentrations of sodium deoxycholate extracted proteins more efficiently from VP8-deficient virions than from wild-type virions. The increased fragility of VP8-deficient virions and their slower RNA extrusion rates suggest that VP8 may be required for the correct formation of the core. Virions which lack VP8 were shown to contain a full complement of transcription enzymes, and soluble extracts from these virions were active in transcription assays using either single-stranded M13 DNA or exogenous plasmid template containing a vaccinia virus early promoter. Thus, the defect in transcription is due not to a lack of specific transcriptional enzymes within virions but rather to the inability of these enzymes to efficiently transcribe the DNA genome packaged within VP8-deficient virions. These results suggest that VP8 is required for the correct packaging of the viral DNA genome and/or for the efficient transcription of packaged virion DNA, which has a higher degree of structural complexity than plasmid templates. Possible roles for VP8 in these processes are discussed.
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