ArticleMolecular and cellular biology1993
Conditional silencing: the HMRE mating-type silencer exerts a rapidly reversible position effect on the yeast HSP82 heat shock gene.
Article in Molecular and cellular biology, 1993. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers.
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Who cites it
26 citing papers in PubMed, 43 citations in OpenAlex.
- Article
- Genetic and epigenetic determinants establish a continuum of Hsf1 occupancy and activity across the yeast genome.Molecular biology of the cell · 2018Article
- Heat Shock Protein Genes Undergo Dynamic Alteration in Their Three-Dimensional Structure and Genome Organization in Response to Thermal Stress.Molecular and cellular biology · 2017Article
- The Nuts and Bolts of Transcriptionally Silent Chromatin in Saccharomyces cerevisiae.Genetics · 2016Review
- Uncoupling transcription from covalent histone modification.PLoS genetics · 2014Article
- Mediator recruitment to heat shock genes requires dual Hsf1 activation domains and mediator tail subunits Med15 and Med16.The Journal of biological chemistry · 2013Article
- Role of Mediator in regulating Pol II elongation and nucleosome displacement in Saccharomyces cerevisiae.Genetics · 2012Article
- p53 Interacts with RNA polymerase II through its core domain and impairs Pol II processivity in vivo.PloS one · 2011Article
- Acetylation of H3 K56 is required for RNA polymerase II transcript elongation through heterochromatin in yeast.Molecular and cellular biology · 2010Article
- SAGA and Rpd3 chromatin modification complexes dynamically regulate heat shock gene structure and expression.The Journal of biological chemistry · 2009Article
- Sir2 silences gene transcription by targeting the transition between RNA polymerase II initiation and elongation.Molecular and cellular biology · 2008Article
- The tumor suppressor p53 associates with gene coding regions and co-traverses with elongating RNA polymerase II in an in vivo model.Oncogene · 2008Article
- A functional module of yeast mediator that governs the dynamic range of heat-shock gene expression.Genetics · 2006Article
- Domain-wide displacement of histones by activated heat shock factor occurs independently of Swi/Snf and is not correlated with RNA polymerase II density.Molecular and cellular biology · 2005Article
- Barrier proteins remodel and modify chromatin to restrict silenced domains.Molecular and cellular biology · 2004Article
- RNA polymerase III and RNA polymerase II promoter complexes are heterochromatin barriers in Saccharomyces cerevisiae.The EMBO journal · 2001Article
- Cell cycle-dependent binding of yeast heat shock factor to nucleosomes.Molecular and cellular biology · 2000Article
- Yeast heterochromatin is a dynamic structure that requires silencers continuously.Genes & development · 2000Article
- UASrpg can function as a heterochromatin boundary element in yeast.Genes & development · 1999Article
- Cooperative binding of heat shock factor to the yeast HSP82 promoter in vivo and in vitro.Molecular and cellular biology · 1999Article
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
Abstract
The HMRE silencer of Saccharomyces cerevisiae has been previously shown to transcriptionally repress class II and class III genes integrated within the HMR silent mating-type locus up to 2.6 kb away. Here we study the ability of this element to repress at an ectopic position, independent of sequences normally associated with it. When integrated 750 bp upstream of the HSP82 heat shock gene, the silencer represses basal-level transcription approximately 5-fold but has no effect on chemical- or heat-shock-induced expression. Such conditional silencing is also seen when the HMRE/HSP82 allele is carried on a centromeric episome or when the entire HMRa domain is transplaced 2.7 kb upstream of HSP82. Notably, the a1 promoter within the immigrant HMRa locus remains fully repressed at the same time HSP82 is derepressed. The position effect mediated by the E silencer is absolutely dependent on the presence of a functional SIR4 gene product, is lost within 1 min following stress induction, and is fully reestablished within 15 min following a return to nonstressful conditions. Similar kinetics of reestablishment are seen in HMRE/HSP82 and HMRa/HSP82 strains, indicating that complete repression can be mediated over thousands of base pairs within minutes. DNase I chromatin mapping reveals that the ABF1, RAP1, and autonomously replicating sequence factor binding sites within the silencer are constitutively occupied in chromatin, unaltered by heat shock or the presence of SIR4. Similarly, the heat shock factor binding site upstream of HSP82 remains occupied under such conditions, suggesting concurrent occupancy of silencer and activator binding sites. Our results are consistent with a model in which silencing at the HMRE/HSP82 allele is mediated by direct or indirect contacts between the silencer protein complex and heat shock factor.
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