ArticleMammalian genome : official journal of the International Mammalian Genome Society1993
Re-localization of Actsk-1 to mouse chromosome 8, a new region of homology with human chromosome 1.
Article in Mammalian genome : official journal of the International Mammalian Genome Society, 1993. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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4 citing papers in PubMed, 6 citations in OpenAlex.
- High-resolution mapping of a linkage group on mouse chromosome 8 conserved on human chromosome 16Q.Mammalian genome : official journal of the International Mammalian Genome Society · 1997Article
- The genes coding for alpha and beta catenin (Catna1 and Catnb) and plakoglobin (Jup) map to mouse chromosomes 18, 9, and 11, respectively.Mammalian genome : official journal of the International Mammalian Genome Society · 1995Article
- Maps from two interspecific backcross DNA panels available as a community genetic mapping resource.Mammalian genome : official journal of the International Mammalian Genome Society · 1994Article
- Linkage of Agt and Actsk-1 to distal mouse chromosome 8 loci: a new conserved linkage.Mammalian genome : official journal of the International Mammalian Genome Society · 1993Article
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Authors and funding
5 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
We present here the genetic mapping of the alpha-skeletal actin locus (Actsk-1) on mouse Chromosome (Chr) 8, on the basis of the PCR analysis of a microsatellite in an interspecific backcross. Linkage and genetic distances were established for four loci by analysis of 192 (or 222) meiotic events and indicated the following gene order: (centromere)-Es-1-11.7 cM-Tat-8.3 cM-Actsk-1-0.5 cM-Aprt. Mapping of ACTSK to human Chr 1 and of TAT and APRT to human Chr 16 demonstrates the existence of a new short region of homology between mouse Chr 8 and human Chr 1. Intermingling on this scale between human and mouse chromosomal homologies that occurred during evolution creates disorders in comparative linkage studies.
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