ReviewThe Journal of clinical investigation1993
Abnormalities in G protein-coupled signal transduction pathways in human disease.
Review in The Journal of clinical investigation, 1993. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
18 citing papers in PubMed.
- Standards of care for hypoparathyroidism in adults: a Canadian and International Consensus.European journal of endocrinology · 2019Review
- Detection of Rare Somatic GNAS Mutation in McCune-Albright Syndrome Using a Novel Peptide Nucleic Acid Probe in a Single Tube.Molecules (Basel, Switzerland) · 2017Article
- Pseudohypoparathyroidism as a rare cause of bilateral slipped capital femoral epiphysis.Indian journal of orthopaedics · 2012Article
- Constitutive activation of G protein-coupled receptors and diseases: insights into mechanisms of activation and therapeutics.Pharmacology & therapeutics · 2008Review
- Alteration in G proteins and prolactin levels in pituitary after ethanol and estrogen treatment.Alcoholism, clinical and experimental research · 2008Article
- Hypothalamic and hypophyseal regulation of growth hormone secretion.Cellular and molecular neurobiology · 1998Review
- Platelet signal transduction defect with Galpha subunit dysfunction and diminished Galphaq in a patient with abnormal platelet responses.Proceedings of the National Academy of Sciences of the United States of America · 1997Article
- Increased proliferation of osteoblastic cells expressing the activating Gs alpha mutation in monostotic and polyostotic fibrous dysplasia.The American journal of pathology · 1997Article
- A 4-base pair deletion mutation of Gs alpha gene in a Japanese patient with pseudohypoparathyroidism.Journal of endocrinological investigation · 1996Article
- G protein gene knockout hits the gut.Nature medicine · 1995Article
- Mutations of signal-transducing G proteins in human disease.Journal of molecular medicine (Berlin, Germany) · 1995Review
- Activation of membrane receptors.Endocrine · 1995Article
- Localization of the gene for a novel human adenylyl cyclase (ADCY7) to chromosome 16.Human genetics · 1995Article
- Arg60 to Leu mutation of the human thromboxane A2 receptor in a dominantly inherited bleeding disorder.The Journal of clinical investigation · 1994Article
- Albright's hereditary osteodystrophy.Journal of medical genetics · 1994Review
- Mechanisms of restenosis.Texas Heart Institute journal · 1994Review
- Regulation of G protein function: implications for heart disease.Molecular and cellular biochemistryReview
- Absence of activating mutations in the hot spots of the LH receptor and Gs-alpha genes in Leydig cell tumors.Journal of endocrinological investigationArticle
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
PubMed holds no abstract for this paper.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.