ArticleDigestive diseases and sciences1994
Excitation-contraction coupling in gastric muscles.
Article in Digestive diseases and sciences, 1994. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
1 citing paper in PubMed.
- The vagus nerve mediates the stomach-brain coherence in rats.NeuroImage · 2022Article
Corrections and comments
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Authors and funding
2 authors.
Funding
Abstract
Several mechanisms contribute to the regulation of force generated by gastric muscles. Phasic contractions in the stomach are triggered by the propagation of electrical slow waves. These events are associated with an influx of Ca2+ and an increase in intracellular Ca2+ sufficient to elicit contraction. Entry of Ca2+ may be supplemented by the release of Ca2+ from intracellular stores. Excitatory agonists enhance the amplitude of slow waves, increase the amplitude of Ca2+ transients, and increase the force of phasic contractions. Inhibitory agonists have opposite effects. Excitatory agonists may also enhance release of Ca2+ from stores via the production of IP3. Excitatory and inhibitory agonists may also regulate the sensitivity of the contractile apparatus for Ca2+ and therefore alter the contractile response to a given change in intracellular Ca2+.
Indexed as
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Registered trials
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