ReviewHuman genetics1995
The genetic contribution to the phenotype.
Review in Human genetics, 1995. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
15 citing papers in PubMed.
- Toward a Symbiotic Perspective on Public Health: Recognizing the Ambivalence of Microbes in the Anthropocene.Microorganisms · 2020Article
- Information handling by the brain: proposal of a new "paradigm" involving the roamer type of volume transmission and the tunneling nanotube type of wiring transmission.Journal of neural transmission (Vienna, Austria : 1996) · 2014Review
- Male-specific repeats in wild Bovidae.Journal of applied genetics · 2012Article
- Use of genetic markers and gene-diet interactions for interrogating population-level causal influences of diet on health.Genes & nutrition · 2011Article
- Mendelian randomization: can genetic epidemiology help redress the failures of observational epidemiology?Human genetics · 2008Review
- The nature and significance of behavioural genetic information.Theoretical medicine and bioethics · 2004Review
- Human gene mutation in pathology and evolution.Journal of inherited metabolic disease · 2002Review
- Can it be a "sin" to understand disease? On "genes" and "eugenics" and an "unconnected connection".Medicine, health care, and philosophy · 2001Review
- Schizophrenia epigenesis?Theoretical medicine and bioethics · 2000Review
- Mechanisms of resistance to pathogenesis in muscular dystrophies.Molecular and cellular biochemistry · 1999Review
- Nonlinearity in the epidemiology of complex health and disease processes.Theoretical medicine and bioethics · 1998Review
- Gene environment interaction.Journal of epidemiology and community health · 1996Review
- Phenylketonuria genotypes correlated to metabolic phenotype groups in Norway.European journal of pediatrics · 1996Article
- Genetic complexity in single gene diseases.BMJ (Clinical research ed.) · 1996Article
- Lysosomal free sialic acid storage disorders with different phenotypic presentations--infantile-form sialic acid storage disease and Salla disease--represent allelic disorders on 6q14-15.American journal of human genetics · 1995Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The phenotype is the result of ontogenetic development. This holds true also at the molecular level, because molecular biological processes take place within the organism. In ontogenesis, genetic and nongenetic factors interact in producing successive states, each of which is the prerequisite, and determines the conditions, for the next one to follow. In this interplay, genes are a necessary, but not sufficient, component. The structures already present, gradients, threshold values, positional relationships, and conditions of the internal milieu, are equally essential. Thus, even monofactorial traits can be considered to be of multifactorial causation, and the varying borderline conditions that arise during development add to the complexity. From this standpoint, it is not to be expected that a mutation has a consistent phenotypic outcome, and the genotype-phenotype relationship may be irregular. In the present review, genotypic heterogeneity versus phenotypic heterogeneity is discussed with the help of some selected examples of hereditary diseases. Conditions and mechanisms contributing to this heterogeneity are addressed. It is concluded that the genotype-phenotype relationship is neither unidimensional, programmatical nor hierarchical in a strict sense. Nevertheless, in particular cases, ontogenetic modification appears to be of minor significance, so that the phenotype of a mutation can be predicted with considerable accuracy. This is no surprise if, depending on the nature of the mutation and the physiological function of the gene affected, the genotype-phenotype relationship is direct. However, this relationship may also be consistent in more complex conditions. It is assumed that the total of the non-genetic influences (epigenetic, environmental) are usually so similar or are compensated by the organism to such an extent that the respective mutation acts as the major variable during ontogenetic development.
Indexed as
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.