Autoantibodies against adhesion molecules and structures in blistering skin diseases.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

13 citing papers in PubMed.

1. Decoding autoimmune disease with single-cell immune repertoire and transcriptome sequencing: mechanisms and therapeutic opportunities.Frontiers in immunology · 2026
   Review
2. From neglect to spotlight: the underappreciated role of B cells in cutaneous inflammatory diseases.Frontiers in immunology · 2024
   Review
3. Apoptolysis: a less understood concept in the pathogenesis of Pemphigus Vulgaris.Apoptosis : an international journal on programmed cell death · 2022
   Review
4. Eph/ephrin signaling in epidermal differentiation and disease.Seminars in cell & developmental biology · 2012
   Review
5. Blockade of autoantibody-initiated tissue damage by using recombinant fab antibody fragments against pathogenic autoantigen.The American journal of pathology · 2010
   Article
6. The Misregulation of Cell Adhesion Components during Tumorigenesis: Overview and Commentary.Journal of oncology · 2010
   Article
7. Molecular mechanisms of glioma cell migration and invasion.Journal of neuro-oncology · 2004
   Review
8. A central role for the armadillo protein plakoglobin in the autoimmune disease pemphigus vulgaris.The Journal of cell biology · 2001
   Article
9. The shed ectodomain of collagen XVII/BP180 is targeted by autoantibodies in different blistering skin diseases.The American journal of pathology · 2000
   Article
10. IgE antibodies in sera from patients with bullous pemphigoid are autoantibodies preferentially directed against the 230-kDa epidermal antigen (BP230)Journal of clinical immunology · 1998
    Article
11. The amino-terminal domain of desmoplakin binds to plakoglobin and clusters desmosomal cadherin-plakoglobin complexes.The Journal of cell biology · 1997
    Article
12. Direct Ca2+-dependent heterophilic interaction between desmosomal cadherins, desmoglein and desmocollin, contributes to cell-cell adhesion.The Journal of cell biology · 1997
    Article
13. Structural basis for major histocompatibility complex (MHC)-linked susceptibility to autoimmunity: charged residues of a single MHC binding pocket confer selective presentation of self-peptides in pemphigus vulgaris.Proceedings of the National Academy of Sciences of the United States of America · 1995
    Article

Corrections and comments

• Comment on
  Autoantibodies to desmoplakin I and II in patients with erythema multiforme.1995

Authors and funding

1 author.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

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