ArticleMolecular and cellular biology1994
A factor induced by differentiation signals in cells of the macrophage lineage binds to the gamma interferon activation site.
Article in Molecular and cellular biology, 1994. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
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Who cites it
11 citing papers in PubMed, 59 citations in OpenAlex.
- Phosphorylation of the Stat1 transactivating domain is required for the response to type I interferons.EMBO reports · 2003Article
- Antiapoptotic activity of Stat5 required during terminal stages of myeloid differentiation.Genes & development · 2000Article
- WRS-85D: A tryptophanyl-tRNA synthetase expressed to high levels in the developing Drosophila salivary gland.Molecular biology of the cell · 1999Article
- Stat1 combines signals derived from IFN-gamma and LPS receptors during macrophage activation.The EMBO journal · 1998Article
- STAT1 pathway is involved in activation of caprine arthritis-encephalitis virus long terminal repeat in monocytes.Journal of virology · 1997Article
- Activation of different Stat5 isoforms contributes to cell-type-restricted signaling in response to interferons.Molecular and cellular biology · 1996Article
- Activation of Stat 5b in erythroid progenitors correlates with the ability of ErbB to induce sustained cell proliferation.Proceedings of the National Academy of Sciences of the United States of America · 1996Article
- Differentiation-regulated serine phosphorylation of STAT1 promotes GAF activation in macrophages.Molecular and cellular biology · 1995Article
- Cloning of murine Stat6 and human Stat6, Stat proteins that are tyrosine phosphorylated in responses to IL-4 and IL-3 but are not required for mitogenesis.Molecular and cellular biology · 1995Article
- Prolactin, growth hormone, erythropoietin and granulocyte-macrophage colony stimulating factor induce MGF-Stat5 DNA binding activity.The EMBO journal · 1995Article
- The signalling pathways of interleukin-6 and gamma interferon converge by the activation of different transcription factors which bind to common responsive DNA elements.Molecular and cellular biology · 1994Article
Corrections and comments
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Authors and funding
6 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Rapid transcriptional induction of genes in response to gamma interferon (IFN-gamma) is mediated by the IFN-gamma activation site (GAS) and its cognate protein, the IFN-gamma activation factor (GAF). We describe a GAS-associated, differentiation-induced factor (DIF) as a potential molecular link between the activities of IFN-gamma and of growth and differentiation factors. DIF DNA binding was activated by colony-stimulating factor 1 in murine macrophages and also during tetradecanoyl phorbol acetate-induced differentiation or IFN-gamma treatment in myeloid U937 cells. IFN-gamma activation of DIF decreased significantly upon monocytic differentiation. DIF binding to DNA was inhibited by antiphosphotyrosine antibodies and could be induced by treatment of U937 cells with vanadate. Unlike GAF, DIF-DNA complexes did not contain the 91-kDa protein (p91) from ISGF-3. DIF bound with high affinity to GAS from the promoters of the IFP 53/tryptophanyl-tRNA synthetase and Fc gamma RI genes, intermediate affinity to the Ly6A/E GAS, and low affinity to the guanylate-binding protein GAS. DIF may belong to a family of cytokine- or growth factor-induced factors binding with variable affinities to GAS-related elements: the interleukin-6-responsive acute-phase response factor associated with GAS from different IFN-inducible promoters but with a different preference of binding compared with DIF. The sis-inducible element of the c-fos promoter bound GAF but not DIF. However, the sis-inducible element could be changed by point mutation to compete for GAF and DIF binding. Our data show DIF to be a novel DNA-binding protein which is activated in response to differentiating signals. Moreover, they suggest that a family of cytokine- or growth factor-regulated proteins integrates and coordinates the responses to cytokines and to growth and differentiation factors by binding to GAS-related elements.
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