Evidence map›Paper›PMID 7504729›Full record

ArticleThe Journal of physiology1993

Modulation of K+ and Ca2+ channels by histamine H1-receptor stimulation in rabbit coronary artery cells.

T Ishikawa, J R Hume, K D Keef

Open access · greenAbstract read
In one paragraph

Article in The Journal of physiology, 1993. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.0field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 68 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Review
  6. KMicrocirculation (New York, N.Y. : 1994) · 2018
    Review
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  8. Review
  9. Review
  10. Article
  11. Article
  12. Effects of pH on vascular tone in rabbit basilar arteries.Journal of Korean medical science · 2004
    Article
  13. Article
  14. Article
  15. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

T IshikawaDepartment of Physiology, University of Nevada School of Medicine, Reno 89557-0046.
J R Hume
K D Keef
University of Nevada, Reno · US

Funding

KINASE REGULATION OF L TYPE CALCIUM CHANNELSR01HL040399 · NHLBI · UNIVERSITY OF NEVADA RENO · PI KEEF, KATHLEEN D · 1991 to 2002
$593k
NEURAL AND METABOLIC CONTROL OF THE CORONARY ARTERYR29HL040399 · NHLBI · UNIVERSITY OF NEVADA RENO · PI KEEF, KATHLEEN D · 1987 to 1989
–
NHLBI NIH HHS HL40399
6 · The paper itself

Abstract

1. The modulation of whole-cell K+ and Ca2+ currents by stimulation of histamine H1-receptors in freshly isolated single smooth muscle cells from the rabbit coronary artery was characterized using the patch-clamp technique at 35 degrees C. Single-channel K+ currents were also analysed using the cell-attached patch configuration. 2. The histamine H1-receptor agonist, 2-(2-aminoethyl)pyridine (AEP) (0.1 mM), increased the amplitude of voltage-activated inward Ba2+ currents, recorded using the perforated-patch recording technique, which could be completely blocked by the dihydropyridine antagonist, nicardipine (1 microM). 3. Whole-cell outward K+ currents in rabbit coronary artery cells could be classified into at least two components: (a) a slowly inactivating, 4-aminopyridine (4-AP)-sensitive low-noise current, and (b) a non-inactivating, tetraethylammonium (TEA)-sensitive high-noise current. 4. AEP (0.1 mM) caused changes in whole-cell outward K+ currents which depended upon membrane voltage. Specifically: (a) AEP enhanced the amplitude of outward currents at voltages between -30 and 0 mV, and (b) AEP decreased the outward currents at more positive potentials. 5. The removal of extracellular Ca2+ caused little inhibition of the effects of AEP on K+ currents, whereas the depletion of intracellular Ca2+ stores by pretreatment with ryanodine and caffeine prevented the effects of AEP on K+ channels. Moreover, acute exposure to ryanodine (10 microM) or thapsigargin (1 microM), a Ca(2+)-ATPase inhibitor, caused voltage-dependent changes in the outward currents similar to those observed with AEP. These results suggest that the voltage-dependent effects of AEP on K+ currents are mainly mediated by release of Ca2+ from intracellular stores. 6. The dual stimulatory and inhibitory effect of AEP on whole-cell K+ currents was shown to be due to a differential effect on two distinct types of K+ channels. The stimulatory effect observed over the voltage range -30 to 0 mV was prevented by pretreatment of cells with low concentrations of TEA (1 mM), whereas the inhibitory effect observed at positive potentials was prevented by pretreatment of cells with 4-AP (3 mM). 7. Single-channel recordings revealed two types of unitary K+ currents with conductances of 225 and 70 pS in the cell-attached configuration with symmetrical K+ solutions (150 mM K+ in pipette-150 mM K+ in bath). Bath application of AEP (0.1 mM) caused a marked increase in the open probability of the large conductance channels.(ABSTRACT TRUNCATED AT 400 WORDS)

Indexed as

4-AminopyridineAnimalsCalciumCalcium ChannelsCharybdotoxinCoronary VesselsHistamine AgonistsIn Vitro TechniquesMalePotassium ChannelsPyridinesRabbitsReceptors, Histamine H1Scorpion VenomsTetraethylammoniumTetraethylammonium Compounds2-(2-aminoethyl)pyridine4-AminopyridineCalciumCalcium ChannelsCharybdotoxinHistamine AgonistsPotassium ChannelsPyridinesReceptors, Histamine H1Scorpion VenomsTetraethylammoniumTetraethylammonium Compounds

Identifiers

PMID7504729
PMCPMC1143832
OpenAlexW2040668875

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.