ArticleJournal of neurology1984
Brain-stem auditory-evoked potentials in post-comatose patients after severe closed head trauma.
Article in Journal of neurology, 1984. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
3 citing papers in PubMed, 21 citations in OpenAlex.
- Electrophysiological Brain Connectivity: Theory and Implementation.IEEE transactions on bio-medical engineering · 2019Article
- Resistor mesh model of a spherical head: part 2: a review of applications to cortical mapping.Medical & biological engineering & computing · 2005Article
- Linear inverse source estimate of combined EEG and MEG data related to voluntary movements.Human brain mapping · 2001Article
Corrections and comments
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Authors and funding
3 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Brain-stem auditory-evoked potentials (BAEPs) were recorded in 35 post-comatose patients after severe closed head trauma. Compared to the normal BAEPs obtained from 30 normal controls, significant individual abnormality could be detected in only 2 patients. This abnormality consisted of lateral asymmetry, but not prolonged interpeak latencies or abnormal amplitude ratios. Further, no differences were found between groups as regards interpeak latencies. A significant reduction of BAEP amplitudes was seen in the patient group, being most pronounced in the 13 patients with hearing loss. It is concluded that the BAEP does not sufficiently reveal brain-stem lesions expected to persist after severe closed head trauma, presumably because the structures generating waves IV and V are not permanently affected. The amplitude reductions observed may be explained by the diffuse cochlear damage known to occur concurrently with head trauma.
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