ArticleResearch in pharmaceutical sciences2026
Article in Research in pharmaceutical sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background and purpose: Hyperlipidemia remains a major cardiovascular risk factor requiring novel therapeutic strategies beyond conventional statin therapy. This study investigated the antihyperlipidemic and antioxidant properties of Experimental approach: Eight chromatographic subfractions were screened for 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase inhibitory activity. Lipid profile, lipid peroxidation, and histopathology were evaluated in rats fed a high-fat diet to induce hyperlipidemia. Chemical composition was characterized using liquid chromatography high-resolution mass spectrometry (LC-HRMS) and subsequently investigated through molecular docking simulations. Findings/Results: Subfraction 4 demonstrated the strongest HMG-CoA reductase inhibitory activity. Conclusion and implications: These findings support the potential of
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.