ReviewThe Yale journal of biology and medicine2026
Toward Understanding the Molecular Basis and Precise Diagnosis of Mitochondrial Diabetes: A Comprehensive Review.
Review in The Yale journal of biology and medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Pathogenic variants in mitochondrial DNA cause mitochondrial dysfunction and a variety of clinical symptoms. Some mtDNA variants are strongly associated with diabetes. Mitochondrial diabetes is primarily caused by β-cell dysfunction and impaired insulin secretion, although insulin resistance may also be present. Mitochondrial dysfunction leads to reduced adenosine triphosphate production in pancreatic β-cells and impaired glucose-stimulated insulin secretion. The most common variant associated with mitochondrial diabetes is m.3243A>G, located in the
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