ArticleMetabolites2026
Triglyceride-Glucose Index and Systemic Inflammatory Markers in Polyendocrine Metabolic Ovarian Syndrome (Polycystic Ovary Syndrome): A Cross-Sectional Study.
Article in Metabolites, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
BACKGROUND/
objectivesLow-grade chronic inflammation is recognized as part of the systemic profile of polyendocrine metabolic ovarian syndrome (PMOS, formerly polycystic ovary syndrome, PCOS), although whether this inflammatory signal tracks insulin resistance, adiposity, or both remains unclear. This study evaluated whether the triglyceride-glucose (TyG) index, a surrogate of insulin resistance, is associated with subclinical inflammation independently of body mass index (BMI) in women with PMOS.
methodsIn this retrospective cross-sectional study of 150 women with PMOS, the TyG index was calculated from fasting triglycerides and glucose. C-reactive protein (CRP; standard assay) and the systemic immune-inflammation index (SII) were pre-specified primary markers; the neutrophil-, platelet-, and monocyte-to-lymphocyte ratios (NLR, PLR, and MLR) and the systemic inflammation response index (SIRI) were secondary. Associations were assessed using Spearman correlation and multivariable linear regression adjusted for age and BMI, with component, extended-adjustment, tertile, and BMI-interaction analyses.
resultsThe TyG index correlated with the homeostatic model assessment of insulin resistance (HOMA-IR) (rho = 0.45,
conclusionsIn PMOS, the TyG index was associated with hemogram-derived inflammatory indices independently of BMI, a signal attributable to its triglyceride and glucose components rather than to fasting insulin, whereas standard-assay CRP tracked adiposity. These cross-sectional findings are hypothesis-generating.
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