Evidence map›Paper›PMID 42918920›Full record

ArticleNeurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology2026

Transient focal neurological episodes in cerebral amyloid angiopathy-related convexity subarachnoid hemorrhage.

Shinya Tomari, Junji Kasuya, Hiroshi Takashima

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Article in Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

3 authors.

Shinya TomariDepartment of Neurology, Atsuchi Neurosurgery Hospital, Kagoshima, Japan. sny5588@gmail.com.ORCID http://orcid.org/0000-0001-9680-1723
Junji KasuyaDepartment of Neurology, Atsuchi Neurosurgery Hospital, Kagoshima, Japan.
Hiroshi TakashimaDepartment of Neurology and Geriatrics, Kagoshima University Graduate School of Medical and Dental Science, Kagoshima, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundConvexity subarachnoid hemorrhage (cSAH) is recognized as a cause of transient focal neurological episodes. These episodes can closely mimic transient ischemic attacks (TIAs). We report a case of cerebral amyloid angiopathy (CAA)-related cSAH presenting with recurrent transient monoparesis. CASE DESCRIPTION: An elderly patient with a history of hypertension was admitted to our hospital because of recurrent episodes of transient left upper limb monoparesis. Initial diffusion-weighted magnetic resonance imaging (DWI) demonstrated a focal hyperintensity over the right frontal convexity. Susceptibility-weighted imaging (SWI) revealed a focal hypointense signal at the corresponding area, consistent with cSAH. Video recordings of the left upper monoparesis were available, but the episodes were clinically indistinguishable from TIAs. Antiseizure treatment with levetiracetam was initiated, but the episodes persisted. After the addition of clobazam, no further episodes were observed.

conclusionThis case demonstrates that CAA-related cSAH may present with recurrent transient monoparesis that closely mimics TIA. Careful evaluation of susceptibility weighted imaging is important for recognizing the underlying hemorrhagic lesion in patients with recurrent stereotyped TFNEs.

Indexed as

Cerebral Amyloid AngiopathyParesisSubarachnoid HemorrhageAgedDiffusion Magnetic Resonance ImagingFemaleHumansIschemic Attack, TransientMagnetic Resonance ImagingMaleConvexity subarachnoid hemorrhageTIA-mimicTransient focal neurological episodes

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.