ArticleCell research2026
Sensory neuron-derived CGRP ameliorates kidney injury and fibrosis via monocytes and macrophages.
Article in Cell research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
10 authors.
Funding
Abstract
Acute kidney injury (AKI) and its progression to chronic kidney disease (CKD) remain major unmet clinical challenges for which effective therapies are lacking. Although sensory neurons are recognized as key regulators of tissue injury and repair via neuroimmune crosstalk, their organization and function within the kidney are poorly understood. Here, we delineate a renal neuroimmune axis in which nociceptive neurons protect against ischemia-reperfusion (I/R)-induced kidney injury. Using whole-organ tissue clearing and three-dimensional imaging, we mapped an extensive network of nociceptive sensory fibers throughout the renal parenchyma. Ablation of TRPV1
Identifiers
42918818What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.