Evidence map›Paper›PMID 42911416›Full record

ArticleHuman genetics2026

The impact of nationwide folic acid fortification on genetic variants associated with conotruncal heart defects.

Sarah U Morton, Enrique Mondragon-Estrada, Rui Qian, Omobola O Oluwafemi, Kit Sing Au, Hope Northrup, Wendy K Chung, A J Agopian, Tina O Findley

Abstract read
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Article in Human genetics, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

9 authors.

Sarah U Morton *Department of Pediatrics, Boston Children's Hospital, Harvard Medical School, Boston, MA, United States. Sarah.Morton@childrens.harvard.edu.ORCID http://orcid.org/0000-0002-7816-2646
Enrique Mondragon-Estrada *Division of Newborn Medicine, Department of Pediatrics, Boston Children's Hospital, Boston, MA, United States.
Rui QianDivision of Newborn Medicine, Department of Pediatrics, Boston Children's Hospital, Boston, MA, United States.
Omobola O OluwafemiDepartment of Epidemiology, UTHealth Houston School of Public Health, Houston, TX, United States.
Kit Sing AuDivision of Medical Genetics, Department of Pediatrics, McGovern Medical School at UTHealth Houston, Houston, TX, United States.
Hope NorthrupDivision of Medical Genetics, Department of Pediatrics, McGovern Medical School at UTHealth Houston, Houston, TX, United States.
Wendy K ChungDepartment of Pediatrics, Boston Children's Hospital, Harvard Medical School, Boston, MA, United States.
A J AgopianDepartment of Epidemiology, UTHealth Houston School of Public Health, Houston, TX, United States.
Tina O FindleyDivision of Neonatal-Perinatal Medicine, Department of Pediatrics, McGovern Medical School at UTHealth Houston, Houston, TX, United States. Tina.O.Findley@uth.tmc.edu.ORCID http://orcid.org/0000-0002-2766-4654

Funding

National Center for Advancing Translational Sciences, National Institutes of Health 5KL2TR003168-02
6 · The paper itself

Abstract

Folate deficiency is associated with increased risk of conotruncal heart defects (CTHD), but its interaction with genetic factors remains unclear. Our objective was to investigate genome-wide associations between common genetic variants and birth before versus after universal folic acid fortification among children with CTHD and other heart defects. Sequencing data were available through the Pediatric Cardiac Genomics Consortium, aligned to the human reference genome (GRCh38/hg38), and jointly processed to ensure uniform variant detection and minimize batch effects. Analyses were restricted to individuals with European-inferred genetic ancestry, limiting generalizability to other ancestral groups. Genome-wide association study (GWAS) models compared allele frequencies across fortification eras among all participants (n = 1285), the subset with CTHD (n = 534), and the remaining individuals with other heart defects (n = 751). Functional enrichment was assessed using the Database for Annotation, Visualization and Integrated Discovery (DAVID). Among the full analytic group, eight loci had at least two suggestive variants before compared to after fortification. Among the subset with CTHD, two variants located in DHRS3 (rs7551703, odds ratio [OR] 2.10, and rs6541043, OR 2.21) and four variants located in PPARGC1B showed suggestive association after fortification (OR 4.47-4.51). Enriched biological pathway terms were consistent with cardiac developmental processes. In summary, this study examined the association between folic acid fortification and genetic risk for CTHD and other heart defects and identified associations with fortification era that suggest some shift in risk following fortification. Identified pathways suggest gene-nutrient interactions may modulate cardiac developmental pathways, underscoring the relationship between maternal folate status and cardiovascular development.

Indexed as

Folic AcidFood, FortifiedHeart Defects, CongenitalChildFemaleGene FrequencyGenetic Predisposition to DiseaseGenetic VariationGenome-Wide Association StudyHumansMalePolymorphism, Single NucleotideFolic Acid

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.