Evidence map›Paper›PMID 42909297›Full record

ReviewFrontiers in immunology2026

Pathogenesis of bronchopulmonary dysplasia: an immunological perspective.

David Riley, Vineet Bhandari

Abstract readReview
In one paragraph

Review in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

David RileyDepartment of Pediatrics, Anne Burnett Marion School of Medicine at Texas Christian University, Fort Worth, TX, United States.
Vineet BhandariDivision of Neonatology, The Children's Regional Hospital at Cooper, Cooper Medical School of Rowan University, Camden, NJ, United States.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Bronchopulmonary dysplasia (BPD), the most common chronic lung disease of prematurity, is at its mechanistic core an immune-developmental disease in which inflammatory injury disrupts normal lung maturation. In the United States, BPD remains among the most common morbidities of very preterm birth, affecting a substantial proportion of infants born before 32 weeks' gestation. These infants are born before alveolarization is complete, into an inflammatory environment that can impair ongoing lung development. This review advances a single thesis: the heterogeneous prenatal and postnatal insults that precede BPD (chorioamnionitis and the fetal inflammatory response syndrome,

Indexed as

Bronchopulmonary DysplasiaLungAnimalsFemaleHumansImmunity, InnateInfant, NewbornInfant, PrematureInflammasomesInnate Immunity RecognitionNLR Family, Pyrin Domain-Containing 3 ProteinPregnancySignal TransductionInflammasomesNLR Family, Pyrin Domain-Containing 3 Proteinbronchopulmonary dysplasiahyperoxiainnate immunitylung developmentmacrophage polarizationNLRP3 inflammasomepreterm infantTLR4

Identifiers

PMID42909297
PMCPMC13684817

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.