Evidence map›Paper›PMID 42908761›Full record

ArticleFrontiers in immunology2026

A transcriptomic atlas of macrophage polarization induced by inflammatory and reparative cytokines reveals cytokine-specific and synergistic polarization programs.

Gregory R Lum, Bria M Gorman, Silvana A Sidhom, Oscar A Davalos, Deepa K Murugesh, Nicole F Leon, Margarita V Rangel, Nicholas R Hum, Aimy Sebastian

Abstract read
In one paragraph

Article in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Gregory R LumLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Bria M GormanLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Silvana A SidhomLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Oscar A DavalosLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Deepa K MurugeshLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Nicole F LeonLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Margarita V RangelLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Nicholas R HumLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.
Aimy SebastianLawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA, United States.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Macrophages are highly plastic immune cells derived from bone marrow progenitors that respond dynamically to environmental cues and coordinate inflammation, debris clearance, tissue remodeling, wound repair, and regeneration. Although traditionally classified into pro-inflammatory (M1) and anti-inflammatory (M2) phenotypes, macrophage activation is now recognized as a continuum of overlapping states shaped by complex signaling environments. However, how diverse microenvironmental cues shape macrophage states and functions remains incompletely defined. Here, we performed a systematic transcriptomic analysis of murine bone marrow-derived macrophages stimulated with a broad panel of inflammatory, regulatory, and reparative cytokines, both individually and in selected combinations. Our analysis identified distinct and overlapping transcriptional programs associated with classical, alternative, and hybrid activation states, and showed that combinatorial cytokine signals modulate these programs beyond the effects of individual stimuli. Notably, combining IL-4 or IL-13 with IL-6 or IL-33 enhanced gene-expression programs associated with tissue repair, angiogenesis, and metabolic reprogramming. Cytokine array analysis further supported distinct, combination-specific secretory responses. Addition of IL-33 to the IL-4 + IL-13 backbone increased secretion of the alternative activation-associated chemokines CCL17 and CCL22, whereas addition of IL-6 uniquely increased secretion of the immunoregulatory cytokine IL-10. The combinations also showed condition-dependent increases in inflammatory mediators, including TNF-α, IL-18, CXCL1, and, to a lesser extent, IL-1β. Together, these findings support distinct mixed secretory programs rather than a uniform macrophage activation state. Overall, this study provides a systematic framework for understanding how individual and combinatorial cytokine cues shape macrophage transcriptional programs. This comprehensive dataset serves as a resource for interpreting macrophage heterogeneity in complex inflammatory and tissue-repair microenvironments and for prioritizing candidate regulatory pathways for future mechanistic investigation.

Indexed as

CytokinesMacrophage ActivationMacrophagesTranscriptomeAnimalsGene Expression ProfilingInflammationMiceMice, Inbred C57BLCytokinescytokine-mediated responsesgene expression profilinginflammation and tissue repairmacrophage activationmacrophage plasticity

Identifiers

PMID42908761
PMCPMC13683169

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.