ArticleSurgical case reports2026
Planned Distal Pancreatectomy and Splenectomy for Pancreatic Tail Necrosis and Splenic Infarction after Embolization for Gastric Ulcer-Induced Splenic Artery Perforation: A Case Report.
Article in Surgical case reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
introductionSplenic artery perforation secondary to gastric ulcer is a rare but life-threatening cause of upper gastrointestinal bleeding. Splenic artery embolization (SAE) can provide rapid hemorrhage control, but delayed ischemic complications involving the pancreatic tail and spleen are uncommon. CASE PRESENTATION: A woman in her 70s presented with hematemesis and profound hemorrhagic shock. Contrast-enhanced CT showed active extravasation from the splenic artery adjacent to the posterior gastric wall. During emergency SAE, coils were deployed across the bleeding point, but persistent extravasation required additional embolization with a 1:2 mixture of N-butyl-2-cyanoacrylate (NBCA) and lipiodol. Final angiography confirmed hemostasis with preservation of the dorsal and great pancreatic arteries. CT on hospital day 2 showed no apparent pancreatic ischemic change. On day 11, newly developed hypoattenuating areas involving the pancreatic tail and part of the spleen were detected, suggestive of ischemic changes. The patient had no abdominal pain or overt clinical deterioration. Laboratory abnormalities that had improved by day 8 showed a secondary increase on day 11, with a white blood cell count of 12200/μL, C-reactive protein level of 3.90 mg/dL, and serum amylase level of 456 U/L. Planned distal pancreatectomy and splenectomy were selected after individualized assessment of the pancreatic and splenic ischemic changes, together with a residual splenic hilar aneurysmal lesion and suspected posterior gastric wall penetration. Pathological examination confirmed pancreatic necrosis and splenic infarction. No postoperative pancreatic fistula or other major postoperative complication occurred, and the patient was discharged on hospital day 38 after rehabilitation.
conclusionsNBCA-assisted SAE can achieve rapid hemostasis in life-threatening splenic arterial bleeding, but post-embolization surveillance remains important. This case illustrates that management of post-SAE ischemic changes should be individualized and that planned surgery in a clinically stable patient should be based on the overall clinical and anatomical context.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.