ArticleJournal of animal science and technology2026
MIT-001 mitigates vitrification-induced mitochondrial stress and regulates filamentous actin reorganization, enhancing the survival and development of vitrified-warmed bovine blastocysts.
Article in Journal of animal science and technology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
1 citing paper in PubMed.
- Differential gene expression in Glisson's capsule associated with liver abscesses in feedlot cattle.Journal of animal science · 2026Article
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Authors and funding
5 authors.
Funding
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Abstract
In this study, we investigated whether MIT-001, a small-molecule reactive oxygen species (ROS) scavenger, improves the re-expansion and viability of bovine blastocysts in response to vitrification-induced mitochondrial dysfunction and stress. Therefore, this study aimed to analyze the protective effects of MIT-001 on the mitochondrial function of bovine blastocysts following vitrification warming. In this experiment, MIT-001 (0.1 μM) was allocated to three culture conditions based on treatment period: (I) warming only (WARM); (II) vitrification only (VITR); and (III) both vitrification and warming (VITR-WARM), compared with the control (Non-treated). Survival analysis of cryopreserved bovine blastocysts revealed that MIT-001 supplementation during the warming period (WARM group) significantly improved (
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