ArticleiScience2026
Alternative splicing of a species- and skeletal muscle-specific microexon diversifies the menu of clathrin heavy chains in mice and rats.
Article in iScience, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Little is known about alternative splicing of microexons in most tissues other than the brain. Humans have two homologous clathrin heavy chains. Ubiquitously expressed, CLTC drives clathrin-mediated endocytosis. Highly expressed in skeletal muscles, CLTCL1 instead regulates glucose metabolism. Previously, we reported that a conserved microexon (exon 31) in the
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