Evidence map›Paper›PMID 42883755›Full record

ArticleInternational journal of molecular sciences2026

SMTP-44D Attenuates Renal Inflammatory/Oxidative Dysfunction in a Mouse Model of Ischemia-Reperfusion-Induced Acute Kidney Injury.

Keita Shibata, Keiji Hasumi, Koji Nobe

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Keita ShibataDepartment of Pharmacology, Graduate School of Pharmacy, Showa University, Tokyo 142-8555, Japan.ORCID 0000-0002-3935-8742
Keiji HasumiDepartment of Applied Biological Science, Tokyo University of Agriculture and Technology, 3-5-8 Saiwaicho, Fuchu, Tokyo 183-8509, Japan.ORCID 0000-0002-3340-7312
Koji NobeDepartment of Pharmacology, Graduate School of Pharmacy, Showa University, Tokyo 142-8555, Japan.

Funding

Japan Society for the Promotion of Science 22K08318Takeda Science Foundation 2024065636
6 · The paper itself

Abstract

Acute kidney injury (AKI) is a critical clinical challenge for which effective pharmacological therapies remain limited. Inflammation and oxidative stress play central roles in its pathogenesis. Here, we investigated the effects of

Indexed as

Acute Kidney InjuryInflammationOxidative StressPhenolsReperfusion InjuryAnimalsAntioxidantsDisease Models, AnimalEpoxide HydrolasesInterleukin-1betaKidneyLipocalin-2MaleMiceMice, Inbred C57BLStachybotrysAntioxidantsEpoxide HydrolasesInterleukin-1betaLipocalin-2PhenolsTumor Necrosis Factor-alphaacute kidney injuryanti-inflammatory activityantioxidant activitySMTP-44D

Identifiers

PMID42883755
PMCPMC13664903

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.