Evidence map›Paper›PMID 42881983›Full record

ArticleJournal of clinical medicine2026

Regression of Anabolic-Androgenic Steroid-Associated Cardiomyopathy Eight Years After Cessation of Anabolic-Androgenic Steroid Abuse: A Case Report and Literature Review.

Pavel S Baranov, Maria V Amosova, Marina N Vukolova, Sergey V Pirozhkov, Yulia A Lutokhina

Abstract readCase Reports
In one paragraph

Article in Journal of clinical medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Pavel S BaranovDepartment of Endocrinology, I.M. Sechenov First Moscow State Medical University (Sechenov University), 119991 Moscow, Russia.ORCID 0009-0003-7210-7392
Maria V AmosovaProfessor's Clinic of Endocrinology and Diabetes LLC, Andreevskaya Naberezhnaya 1, Bldg. 1, 119334 Moscow, Russia.ORCID 0000-0003-1848-8721
Marina N VukolovaDepartment of Pathological Physiology, I.M. Sechenov First Moscow State Medical University (Sechenov University), 119991 Moscow, Russia.ORCID 0000-0002-9046-169X
Sergey V PirozhkovDepartment of Pathological Physiology, I.M. Sechenov First Moscow State Medical University (Sechenov University), 119991 Moscow, Russia.ORCID 0000-0002-7116-3398
Yulia A LutokhinaDepartment of Internal Medicine No. 1, I.M. Sechenov First Moscow State Medical University (Sechenov University), 119991 Moscow, Russia.ORCID 0000-0002-7154-6794

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The non-medical use of anabolic-androgenic steroids (AAS) is widespread among strength-training athletes and, increasingly, among men who have no medical indication for testosterone therapy. The cardiotoxic profile of these drugs includes concentric myocardial hypertrophy, systolic and diastolic dysfunction, myocardial fibrosis and an elevated risk of sudden cardiac death. Whether AAS-associated cardiomyopathy is reversible once AAS are withdrawn remains unsettled: published outcomes range from near-complete recovery to irreversible severe biventricular dysfunction. Here we report the case of a man first assessed at the age of 26, after 33 cumulative months of supraphysiological AAS abuse, and reassessed eight years later. Marked regression of concentric left ventricular hypertrophy, with an accompanying reduction in right ventricular free wall thickness, was documented: left ventricular (LV) mass index fell from 261 to 111 g/m

Indexed as

AAS-associated cardiomyopathyanabolic-androgenic steroidsbodybuildingcase reportechocardiographyleft ventricular hypertrophyreverse remodeling

Identifiers

PMID42881983
PMCPMC13665845

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.