ReviewBiology direct2026
Redox imbalance in urology: mechanistic insights into the development and progression of selected urological conditions.
Review in Biology direct, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
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0 citing papers in PubMed.
No citing paper in PubMed yet.
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Reactive oxygen species (ROS) are key mediators of cellular signalling and play a vital role in regulating both physiological and pathological processes. In urologically relevant tissues, particularly the prostate, bladder and kidneys, redox processes influence vascular function, immune responses, cell proliferation, metabolic adaptations and programmed cell death. A disruption of redox homeostasis contributes significantly to the development and progression of urological diseases, including prostate cancer, urothelial carcinoma of the bladder and renal cell carcinoma. This review article examines the mechanisms underlying the transition from physiological redox regulation to pathological dysregulation, as well as their significance for tumourigenesis, the tumour microenvironment, therapeutic resistance and disease progression. Finally, diagnostic and therapeutic perspectives of redox-based strategies, including innovative approaches such as medical gas plasma, are discussed.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.