Evidence map›Paper›PMID 42876811›Full record

ArticleFrontiers in cell and developmental biology2026

G3BP1 reduction during cellular senescence promotes SVIL expression and selected senescence-associated phenotypes.

Hyeong Bin Jeon, Dahyeon Koh, Dohyang Kim, Chaehwan Oh, Yebin Lee, Minjung Lim, Sanghoon Lee, Ji Yun Han, Jae Hong Yoo, Kyung-Seok Han and 4 more

Abstract read
In one paragraph

Article in Frontiers in cell and developmental biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors.

Hyeong Bin JeonDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Dahyeon KohDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Dohyang KimDepartment of Physiology, Daegu Catholic University School of Medicine, Daegu, Republic of Korea.
Chaehwan OhDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Yebin LeeDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Minjung LimDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Sanghoon LeeDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Ji Yun HanDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Jae Hong YooDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Kyung-Seok HanDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.
Myriam GorospeLaboratory of Genetics and Genomics, National Institute on Aging Intramural Research Program, National Institutes of Health, Baltimore, MD, United States.
Jaewoo HongDepartment of Physiology, Daegu Catholic University School of Medicine, Daegu, Republic of Korea.
Ji Heon NohMolecular Aging Biology Laboratory (MABL), Department of Biochemistry, College of Natural Science, Chungnam National University, Daejeon, Republic of Korea.
Kyoung Mi KimDepartment of Biological Sciences, Chungnam National University, Daejeon, Republic of Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

RNA-binding proteins (RBPs) can regulate gene expression through multiple post-transcriptional mechanisms, including the control of mRNA stability. In this study, we identified the Ras GTPase-activating protein-binding protein 1 (G3BP1) as a post-transcriptional regulator of

Indexed as

cellular senescencepost-transcriptional regulationRas GTPase-activating protein-binding protein 1senescence-associated secretory phenotypesupervillin

Identifiers

PMID42876811
PMCPMC13661574

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.