ArticleFrontiers in immunology2026
A defined isothiocyanate mixture suppresses platelet signaling and metabolism to limit thrombosis
Article in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Pathological platelet activation drives thrombosis and is a major contributor to morbidity and mortality. Current antiplatelet therapies reduce thrombotic risk but target a limited range of signaling pathways, have minimal impact on established platelet aggregates, and are frequently associated with bleeding complications. These drawbacks have prompted interest in alternative antiplatelet strategies. Isothiocyanates (ITCs), phytochemicals from cruciferous plants, have been reported to inhibit platelet function; however, their mechanisms and translational relevance remain incompletely defined. The effects of a defined mixture of allyl-, benzyl-, and phenethyl-isothiocyanate on platelet function were evaluated using
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