ArticleThe Plant journal : for cell and molecular biology2026
The RcDREB1E-RcMYB28 module improves drought tolerance in Rosa chinensis via RcLAR/RcCORA1-mediated proanthocyanidin biosynthesis and stress responses.
Article in The Plant journal : for cell and molecular biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
9 authors.
Funding
Abstract
Drought is widely recognized as one of the most severe abiotic stresses limiting plant growth and productivity. Proanthocyanidins (PAs), as key functional secondary metabolites, are central to enhancing plant tolerance to abiotic stresses. Nevertheless, the specific mechanisms underlying PA-mediated drought resistance in rose remain largely unclear. Herein, we demonstrate that drought-induced PA accumulation constitutes a conserved evolutionary mechanism across the plant kingdom, ranging from unicellular algae to angiosperms. Specifically, according to the drought-responsive transcriptome dataset of Rosa chinensis 'Old Blush', an R2R3-MYB transcription factor, RcMYB28, was identified. Overexpression of RcMYB28 remarkably enhanced drought tolerance and induced a significant increase in PAs accumulation, whereas plants with silenced RcMYB28 displayed the opposite phenotypic characteristics. Specifically, RcMYB28 directly regulated the expression of leucoanthocyanidin reductase (RcLAR) and a cold and drought regulatory-protein encoding gene (RcCORA1), thereby promoting PA synthesis and enhancing the stress response. Moreover, we identified RcDREB1E as a potential upstream transcriptional regulator of RcMYB28. RcDREB1E exerts a positive regulatory effect on drought tolerance and PAs accumulation via RcMYB28 expression. Collectively, our results demonstrate a new transcriptional regulatory module (RcDREB1E-RcMYB28) underlying drought-triggered PAs accumulation, which offers valuable genetic resources for engineering enhanced drought tolerance in rose.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.