ArticleRedox biology2026

Cardiomyocyte-specific deletion of thioredoxin promotes myocardial injury, infarction and mitochondrial dysfunction: translocation of cytosolic thioredoxin into mitochondria rescue myocardial dysfunction by restoration of cardiac mitochondrial structure and function.

Sudhir Kumar et al.PubMed ↗Full text ↗Publisher ↗

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