Evidence map›Paper›PMID 42867534›Full record

ArticleActa neuropathologica2026

Incident versus prevalent progressive supranuclear palsy: analyses of U.S. Alzheimer's Disease Research Centers data indicate clinical and neuropathological features of a relatively common and underrecognized tauopathy in the aging brain.

Benjamin J Cushing, Ryan K Shahidehpour, Erin L Abner, Gregory A Jicha, Janna M Neltner, Madeline K Breig, Kailen L Teodorescu, Allison M Neltner, E Pinar Coskun, Gabor G Kovacs and 1 more

Abstract read
In one paragraph

Article in Acta neuropathologica, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0cells of the map it votes in
0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Benjamin J Cushing *Department of Pathology and Laboratory Medicine, University of Kentucky, Lexington, USA.
Ryan K ShahidehpourSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
Erin L AbnerSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
Gregory A JichaSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
Janna M NeltnerDepartment of Pathology and Laboratory Medicine, University of Kentucky, Lexington, USA.
Madeline K BreigSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
Kailen L TeodorescuSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
Allison M NeltnerSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
E Pinar CoskunSanders-Brown Center on Aging, University of Kentucky, 800 S. Limestone Ave, Lexington, KY, 40536-0230, USA.
Gabor G KovacsDepartment of Laboratory Medicine and Pathobiology at the University of Toronto, Toronto, Canada.
Peter T NelsonDepartment of Pathology and Laboratory Medicine, University of Kentucky, Lexington, USA. peter.nelson@uky.edu.

Funding

Strategies for Targeting Astrocyte Reactivity in Alzheimer's Disease and Related DementiasP01AG078116 · NIA · UNIVERSITY OF KENTUCKY · PI PETER T. NELSON · 2022 to 2026
$25.0M
University of Kentucky Alzheimer's Disease Research CenterP30AG072946 · NIA · UNIVERSITY OF KENTUCKY · PI LINDA J VAN ELDIK · 2021 to 2026
$23.5M
Development of novel genomic approaches for profiling cellular temporal-spatial dynamics of neurogenesis in Aging and Alzheimer's diseaseR01AG076932 · NIA · ROCKEFELLER UNIVERSITY · PI Junyue Cao · 2022 to 2026
$4.0M
Genetic Architecture of Aging-Related TDP-43 and Mixed Pathology DementiaRF1AG082339 · NIA · UNIVERSITY OF KENTUCKY · PI FARDO, DAVID WILLIAM, NELSON, PETER T. · 2023 to 2023
$1.7M
Age-related TDP-43 neuropathology: using disease-driving mechanisms to guide classificationR01NS118584 · NINDS · METHODIST HOSPITAL RESEARCH INSTITUTE · PI CYKOWSKI, MATTHEW DANIEL, NELSON, PETER T. · 2024 to 2024
$791k
NIA NIH HHS P01 AG078116NIA NIH HHS P30 AG072946NIA NIH HHS R01 AG076932NIA NIH HHS RF1 AG082339NIH P30 AG072946, R01 NS118584, RF1 AG082339, P01 AG078116, and R01 AG076932NINDS NIH HHS R01 NS118584
6 · The paper itself

Abstract

Progressive supranuclear palsy (PSP) is an adult-onset neurodegenerative disease with a heterogeneous spectrum of clinical and pathological features. The epidemiology of PSP-as defined by the gold standard of autopsy diagnosis-remains unresolved, but prevalence estimates are typically ~1:10,000. Here, we analyzed the National Alzheimer's Coordinating Center (NACC) Neuropathology Data Set, aggregated from 38 U.S. Alzheimer's Disease Research Centers (ADRCs). We compared clinical and neuropathologic features of autopsied participants, stratifying on cognitive status at recruitment into the cohort. The sample comprised 6845 individuals who were followed longitudinally for 4.0 (± 2.9 stdev) approximately yearly visits, n = 297 with eventual autopsy-verified PSP. Participants were not necessarily diagnosed or followed in movement disorder clinics. Among those with dementia or PSP at recruitment (n = 4310), 3.3% had corticobasal degeneration (CBD), 3.0% Pick's disease, and 4.5% PSP pathology. Among those recruited while cognitively normal (n = 1451), 0.7% had CBD, and 0.1% Pick's disease, while 3.2% had PSP pathology. This frequency of incident PSP pathology was higher than expected based on prior epidemiology studies. In comparing between incident (recruited normal) and prevalent (recruited with dementia or clinical PSP) cases with PSP pathology, those with incident PSP died older: 89.5 years vs 76.3 years on average. Furthermore, incident PSP pathology cases were less likely to manifest stereotypical PSP clinical features, but more likely to have parkinsonism. In a convenience sample of individuals with PSP pathology from the University of Kentucky ADRC (n = 23), digital pathology analyses using HALO software and AI-based analytic modules revealed that PSP tau pathology was more severe overall in prevalent PSP. Within the putamen, incident cases had a higher proportion of tufted astrocytes and lower proportion of NFTs. In conclusion, in this large multicenter sample, PSP pathology was more common than other FTLD-Tau subtypes, and incident PSP often differed clinically from "classic" PSP.

Indexed as

AgingAlzheimer DiseaseBrainSupranuclear Palsy, ProgressiveTauopathiesAgedAged, 80 and overFemaleHumansIncidenceLongitudinal StudiesMalePrevalencetau ProteinsUnited Statestau Proteins4R tauopathyAGDFTDIncidenceMAPTScanScope

Identifiers

PMID42867534
PMCPMC13661695

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.