Reduced dorsal CA1 activity limits retention of the temporal component of declarative memory in the

Elise Caitlin Rawlinson, Michael Hugh McCullough, Azza Sellami, Aline Marighetto, Alice Shaam Al-Abed, Nathalie Dehorter

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Article in eLife, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it

0cells of the map it votes in

0citing papers in PubMed

–field-weighted citation impact

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Elise Caitlin RawlinsonEccles Institute of Neuroscience, John Curtin School of Medical Research, The Australian National University, Canberra, Australia.ORCID https://orcid.org/0009-0001-5915-8019

Michael Hugh McCulloughEccles Institute of Neuroscience, John Curtin School of Medical Research, The Australian National University, Canberra, Australia.ORCID https://orcid.org/0000-0001-6572-565X

Azza SellamiNeurocentre Magendie, Physiopathologie de la plasticité neuronale, U1215, INSERM, Bordeaux, France.ORCID https://orcid.org/0000-0003-4033-9212

Aline MarighettoNeurocentre Magendie, Physiopathologie de la plasticité neuronale, U1215, INSERM, Bordeaux, France.ORCID https://orcid.org/0000-0002-8893-278X

Alice Shaam Al-Abed *Eccles Institute of Neuroscience, John Curtin School of Medical Research, The Australian National University, Canberra, Australia.ORCID https://orcid.org/0000-0002-6091-810X

Nathalie Dehorter *Eccles Institute of Neuroscience, John Curtin School of Medical Research, The Australian National University, Canberra, Australia.ORCID https://orcid.org/0000-0001-7599-2866

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Growing evidence implicates the hippocampus in the pathophysiology of autism spectrum disorder, particularly in the domains of social interactions and cognition. Yet, the mechanisms driving the hippocampal-dependent cognitive atypicalities in autism remain poorly defined. Here, we characterized how dysfunction of the CA1 subfield of the dorsal hippocampus drives abnormalities in critical components of declarative memory. Using trace fear conditioning in the

Indexed as

Autistic DisorderCA1 Region, HippocampalMembrane ProteinsMemoryNerve Tissue ProteinsAnimalsDisease Models, AnimalFearMaleMiceMice, KnockoutCNTNAP2 protein, mouseMembrane ProteinsNerve Tissue Proteinsautismdeclarative memoryhippocampusmouseneuroscience

Identifiers

PMID42867479

DOI10.7554/eLife.111851

PMCPMC13661045

What OpenQuestion holds

Textmetadata

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.