ArticleThe Journal of antibiotics2026
Inhibition of vasculogenic mimicry by 22-benzyloxydaphlongaminone via glycolysis suppression.
Article in The Journal of antibiotics, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Vasculogenic mimicry (VM) is a phenomenon in which tumor cells themselves form vascular-like tubular structures within the tumor. Identification of novel VM inhibitors is an important research subject in tumor biology. In this study, we focused on 22-benzyloxydaphlongaminone (22-BDO), a synthetic intermediate of isodaphlongamine H. We investigated its effects on VM formation and the underlying mechanism of action. We found that 22-BDO treatment suppressed VM formation in lung adenocarcinoma A549 cells. ChemProteoBase analysis revealed decreased expression of the glycolytic enzymes aldolase A and enolase 1 following 22-BDO treatment, with glycolysis-related proteins showing an overall downward trend. In contrast, no clear similarity to known compounds was observed, suggesting that 22-BDO has a novel mode of action. Furthermore, 22-BDO reduced intracellular ATP levels, and the inhibitory activity correlated with the antiproliferative activity observed for isodaphlongamine H and its synthetic intermediates. Cell death induced by 22-BDO was partially suppressed by the addition of sodium pyruvate. In addition, 2-deoxy-D-glucose also suppressed VM formation, suggesting that energy metabolic pathways, including glycolysis, are involved in VM formation. Taken together, these findings identify 22-BDO as a novel inhibitor of VM formation and suggest an association between metabolic alterations and VM suppression.
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