Evidence map›Paper›PMID 42848827›Full record

ArticlePLoS pathogens2026

A pore-forming toxin monalysin contributes to infection-induced suppression of defecation in female Drosophila.

Marko Rubinić, Yi Yu, Kathirvel Alagesan, Dagmar Frahm, Igor Iatsenko

Abstract read
In one paragraph

Article in PLoS pathogens, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Marko RubinićResearch group Genetics of host-microbe interactions, Max Planck Institute for Infection Biology, Berlin, Germany.ORCID https://orcid.org/0009-0005-0381-6499
Yi YuResearch group Genetics of host-microbe interactions, Max Planck Institute for Infection Biology, Berlin, Germany.ORCID https://orcid.org/0000-0002-9061-0405
Kathirvel AlagesanMax Planck Unit for the Science of Pathogens, Berlin, Germany.
Dagmar FrahmResearch group Genetics of host-microbe interactions, Max Planck Institute for Infection Biology, Berlin, Germany.
Igor IatsenkoResearch group Genetics of host-microbe interactions, Max Planck Institute for Infection Biology, Berlin, Germany.ORCID https://orcid.org/0000-0002-9249-8998

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Pathogen expulsion from the gut via defecation is an important defence strategy against infection. The microbial factors that can subvert this defence reaction remain poorly understood. While many microbes have been found to increase host intestinal peristalsis, Pseudomonas entomophila infection in Drosophila melanogaster leads to infection-induced defecation blockage, particularly in females. Here, we show that this phenotype is driven by a secreted, thermosensitive protein regulated by the GacS/GacA two-component system. Proteomic comparison of the ΔgacA mutant, which only partially inhibits defecation, and the avirulent Δhfq mutant lacking RNA chaperon Hfq, which still triggers the phenotype, identified pore-forming toxin Monalysin as one of the candidate factors required for inhibiting defecation. Consistent with these findings, the Monalysin-deficient mutant failed to fully suppress defecation and exhibited attenuated virulence and reduced persistence in the gut. Hence, Monalysin besides causing intestinal damage, has a previously unknown role in suppressing defecation and pathogen expulsion. Overall, our study identified a bacterial factor that rapidly reduces defecation frequency, consistent with transient suppression of intestinal transit, thus advancing our understanding of pathogen strategies used to subvert host defences.

Indexed as

Bacterial ProteinsBacterial ToxinsDefecationDrosophila melanogasterPseudomonasPseudomonas InfectionsAnimalsFemaleBacterial ProteinsBacterial Toxins

Identifiers

PMID42848827
PMCPMC13671641

What OpenQuestion holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.