ArticleEuropean journal of immunology2026
PGE2 Enhances Human ILC3 Function but Constrains ILC2-to-ILC3 Plasticity.
Article in European journal of immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Type 2 innate lymphoid cells (ILC2) drive eosinophilic asthma, whereas ILC3 and plastic ILC2/3 states are implicated in neutrophilic and mixed granulocytic disease endotypes. While lipid mediators regulate ILC2 function and airway inflammation, their role in human ILC3 biology and ILC plasticity remains poorly defined. We investigated how eicosanoid biosynthesis and signalling regulate human ILC2 and ILC3 function across canonical and plastic states. Here, we identified distinct prostaglandin (PG) profiles across ILC subsets. Unlike ILC2, which produce and rely on PGD
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