ArticleFrontiers in cell and developmental biology2026
From retina to brain: retinal dysfunction and dopaminergic neurodegeneration in
Article in Frontiers in cell and developmental biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Parkinson's disease (PD) is characterized by the progressive loss of dopaminergic neurons within the substantia nigra pars compacta. Although PD is primarily defined as a motor disorder, prodromal non-motor symptoms - such as circadian clock desynchronization and sleep disturbances - can emerge years before clinical diagnosis. Increasing evidence suggests that these non-motor manifestations, particularly circadian disruption, may contribute to disease progression. However, it remains unclear whether circadian dysfunction actively drives neurodegeneration or arises as a consequence of ongoing neuronal loss. In this study, we employed genetic models of PD in
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.